Endometriosis: A Comprehensive and Concise Review of Potential Biomarkers and Treatment Therapies

Namra Aziz, Pranay Wal, Chandana Pyne, Shweta Singh, Sudarshan N. Borkar, Swarnima Pandey, Mukesh C. Sharma
In: Current Drug Therapy · 2024 · vol. 20(7) , pp. 955–971 · doi:10.2174/0115748855310120240819062459 · W4401894409
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⚙ AI-generated summary by claude@2026-06, 2026-06-07 ⓘ

This review explores recent advancements in endometriosis biomarkers and treatment therapies, based on a comprehensive literature analysis of this hormone-dependent inflammatory gynecological illness.

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Abstract

Females of reproductive age can develop endometriosis, an inflammatory gynecological illness that is hormone-dependent. The variable lesions that make up the functional endometrium outside of the uterus cavity are what set this disorder apart in terms of its clinical and pathological aspects. Around 10% of women in their reproductive years have endometriosis worldwide, which significantly lowers their quality of life overall. Infertility and chronic pelvic discomfort are the two main symptoms that significantly affect women's reproductive health and general well-being. The pathophysiological underpinnings of endometriosis remain unclear despite years of intensive research. Understanding the fundamental mechanisms that produce endometriosis is crucial, as demonstrated by the substantial public and private health consequences. This article offers an overview of recent advancements in biomarkers regarding endometriosis treatment options, based on a thorough literature study.

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Condition tags

endometriosisinfertility

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (100)

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last seen: 2026-06-04T00:00:01.174412+00:00
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