Naringenin ameliorates progression of endometriosis by modulating Nrf2/Keap1/HO1 axis and inducing apoptosis in rats

article OA: closed CC0 ⤵ 34 in-corpus citations
Limited metadata. Only one source feed has indexed this record so far — no abstract, full text, or open-access copy is available through Endo Lab. The publisher's page (linked below) is the canonical location for the actual content. If you have institutional access, use "Find at my library".
AI-generated summary by claude@2026-06, 2026-06-06

Naringenin treatment in rats reduced endometriosis progression by activating the Nrf2/Keap1/HO1 pathway and promoting apoptotic cell death.

One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works

My notes (saved in your browser only)

Condition tags

mesh:D004715endometriosis

MeSH descriptors

Apoptosis Endometriosis Flavanones Heme Oxygenase (Decyclizing) Kelch-Like ECH-Associated Protein 1 NF-E2-Related Factor 2 Animals Biomarkers Biomarkers Cell Line, Tumor Cell Movement Disease Progression Endometriosis Endometrium Endometrium Female Flavanones Heme Oxygenase (Decyclizing) Kelch-Like ECH-Associated Protein 1 Membrane Potential, Mitochondrial

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (55)

Cited by (34)

Source provenance

europepmc
last seen: 2026-06-04T01:30:01.192114+00:00
openalex
last seen: 2026-06-10T17:14:06.276822+00:00
pubmed
last seen: 2026-05-13T22:22:41.077124+00:00
License: CC0 · commercial use OK