Atherosclerosis, Oxidation and Endometriosis

Nalini Santanam, Mingqing Song, Song M, Rong R, Rong Rong, Ana A. Murphy, Murphy AA, Sampath Parthasarathy, Parthasarathy S
Free radical research · 2002 · vol. 36(12) , pp. 1315–1321 · doi:10.1080/1071576021000049908 · PMID:12607823 · W2007812459
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This review proposes that oxidative stress and oxidized LDL contribute to both atherosclerosis and endometriosis, diseases characterized by activated macrophages and inflammation.

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Abstract

Endometriosis affects younger women of childbearing age. Atherosclerosis is considered as a disease of the old and increases with the ageing process. Both diseases are characterized by the increased presence of activated macrophages and associated increases in growth promoting activity and the production of inflammatory cytokines. In this review, we propose that oxidative stress and the presence of forms of oxidized low-density lipoprotein (LDL) might contribute to both Atherosclerosis and Endometriosis.

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Condition tags

endometriosis

MeSH descriptors

Aging Arteriosclerosis Endometriosis Oxygen Animals Arteriosclerosis Cell Adhesion Cytokines Cytokines Endometriosis Female Humans Lipoproteins Lipoproteins Lipoproteins, LDL Lipoproteins, LDL Macrophages Macrophages Oxidative Stress Oxygen

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (53)

Cited by (37)

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License: CC0 · commercial use OK