Endometriosis as a model for inflammation–hormone interactions in ovarian and breast cancers
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This paper proposes endometriosis as a model to study how inflammation and hormone interactions contribute to the development of ovarian and breast cancers.
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References (100)
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Cited by (30)
- Biomarkers of endometriosis 2025
- Dissecting the cell microenvironment of ovarian endometrioma through single-cell RNA sequencing 2024
- Markedly increased risk of malignancies in women with endometriosis 2021
- A Nationwide Cohort Study on the risk of non‐gynecological cancers in women with surgically verified endometriosis 2018
- BAF250a Expression in Atypical Endometriosis and Endometriosis-Associated Ovarian Cancer 2016
- Effect of Wnt/β-catenin signal pathway on of matrix metalloproteinase-7 and vascular endothelial growth factor gene expressions in endometriosis 2016
- Endometriosis Located Proximal to or Remote From the Uterus Differentially Affects Uterine Gene Expression 2015
- Complement Pathway Is Frequently Altered in Endometriosis and Endometriosis-Associated Ovarian Cancer 2014
- Decidualisation of ovarian endometriomas in pregnancy: a management dilemma. A case report and review of the literature 2014
- Inflammation and ovarian cancer – current views 2013
- Women with endometriosis have a higher DNA repair capacity and diminished breast cancer risk. 2013
- Endometriosis-Associated Ovarian Cancer: A Review of Pathogenesis 2013
- Endometriosis and ovarian cancer 2013
- Down-regulation of 8-hydroxydeoxyguanosine and peroxiredoxin II in the pathogenesis of endometriosis-associated ovarian cancer. 2012
- Endometriosis 2011
- Basal and Steroid Hormone-Regulated Expression of CXCR4 in Human Endometrium and Endometriosis 2010
- Endometriosis and ovarian cancer: A review 2010
- Ovarian cancer in endometriosis: molecular biology, pathology, and clinical management 2009
- Commentary: Endometriosis--epidemiologic considerations for a potentially 'high-risk' population 2009
- Endometriosis and ovarian cancer: A review 2009
- ERB-041, a selective ERβ agonist, inhibits iNOS production in LPS-activated peritoneal macrophages of endometriosis via suppression of NF-κB activation 2009
- Antioxidant enzymes and lipid peroxidation in endometrium of patients with polyps, myoma, hyperplasia and adenocarcinoma 2009
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