[Research progress on oxidative stress in pathogenesis of endometriosis].

Jiayi He, Xinmei Zhang
Zhejiang da xue xue bao. Yi xue ban = Journal of Zhejiang University. Medical sciences · 2018 · vol. 47(4) , pp. 419–425 · doi:10.3785/j.issn.1008-9292.2018.08.15 · PMID:30511531 · W2945345028
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⚙ AI-generated summary by claude@2026-06, 2026-06-07 ⓘ

This review analyzes how increased oxide production and decreased antioxidant capacity, driven by factors like mitochondrial dysfunction and iron overload, contribute to the pathogenesis of endometriosis.

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⚙ AI-generated deep summary by claude@2026-06, 2026-06-07 · read from full text ⓘ

This article is a research-progress review examining oxidative stress as a mechanism involved in the pathogenesis of endometriosis, synthesizing prior findings across studies. It describes how oxidative stress–related molecular and cellular changes are thought to contribute to disease development and progression, highlighting patterns reported in the literature. The paper’s limitation is that, as a review, it does not present new experimental data and depends on the quality and heterogeneity of included studies. This paper is centrally about endometriosis — specifically, it reviews the role of oxidative stress in endometriosis pathogenesis.

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Abstract

A large number of studies have shown that the oxidative imbalance is common in patients with endometriosis. Abnormal respiratory chain of mitochondrial, estrogen metabolism imbalance, iron overload, and ectopic foci may increase active oxygen, reduction of antioxidant enzyme and non-enzymatic substances may result in decreased antioxidant level, and the exposure to environmental hazards may further aggravate oxidative imbalance in patients with endometriosis. This article analyzes the oxidative imbalance and its role in the pathogenesis of endometriosis from the aspects of excessive oxide production and decreased antioxidant capacity.
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Condition tags

mesh:D004715endometriosis

MeSH descriptors

Endometriosis Endometriosis Oxidative Stress Antioxidants Antioxidants Female Humans Reactive Oxygen Species Reactive Oxygen Species

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (56)

Cited by (1)

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