Lymphocyte subsets in endometriosis

Obstetrics and gynecology · 1984 · vol. 63(4) , pp. 463–6 · PMID:6230549 · W2422317586
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⚙ AI-generated summary by gemini-2.5-flash-lite, 2026-06-07 ⓘ

This study found no statistical differences in lymphocyte subsets or total lymphocyte numbers between women with endometriosis and healthy controls, suggesting numeric differences in lymphocyte subpopulations are unlikely to contribute to an immunologic etiology of endometriosis.

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Abstract

Recent investigations suggested a possible immunologic etiology for endometriosis. To determine whether cellular immunity may be affected in patients with endometriosis, peripheral lymphocyte populations were investigated in 31 confirmed endometriosis patients and 22 control patients. No statistical differences in OKT3, OKT4, OKT8, OKM1, and OKTa1 cells could be observed. Total lymphocyte numbers were within normal ranges for both groups. Helper/suppressor ratios were not statistically different and were in a normal range for both groups. It is concluded that numeric differences in lymphocyte subpopulations represent only a very unlikely contributing factor in a potential immunologic etiology of endometriosis.

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Condition tags

endometriosis

MeSH descriptors

Antibodies, Monoclonal Endometriosis T-Lymphocytes Antibodies, Monoclonal Antibodies, Monoclonal B-Lymphocytes B-Lymphocytes B-Lymphocytes Endometriosis Endometriosis Female Female Humans Humans Immunity, Cellular Immunity, Cellular Leukocyte Count Leukocyte Count Lymphocytes, Null Lymphocytes, Null

Citation neighborhood (2-hop)

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. Outer rings show 2-hop neighbours — papers reached through the immediate citers/citees. [ collapse to 1-hop ]

Cited by (39)

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