Prolonged stimulation with tumor necrosis factor-α induced partial methylation at PR-B promoter in immortalized epithelial-like endometriotic cells

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Tumor necrosis factor-α prolonged stimulation induced partial methylation at the PR-B promoter in immortalized epithelial-like endometriotic cells.

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Abstract

The promoter of P receptor isoform B (PR-B) has recently been shown to be hypermethylated in endometriotic tissues, but one question remains as what causes the aberrant methylation. We show that sustained stimulation of an epithelial-like endometriotic cell line with tumor necrosis factor alpha stimulation for 30 days induced partial methylation in the promoter region of PR-B with concomitant reduction of PR-B expression.

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Condition tags

endometriosis

MeSH descriptors

DNA Methylation Endometriosis Endometrium Epithelial Cells Promoter Regions, Genetic Receptors, Progesterone Tumor Necrosis Factor-alpha Cell Line Down-Regulation Endometriosis Endometriosis Endometrium Epithelial Cells Female Humans Receptors, Progesterone Receptors, Progesterone Stromal Cells Stromal Cells Time Factors

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Cited by (50)

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europepmc
last seen: 2026-08-16T09:21:09.727480+00:00
openalex
last seen: 2026-06-10T17:14:06.276822+00:00
pubmed
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