Endometriosis and Epigenetics: What do we Know?
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This review explores the multiplicity of epigenetic changes associated with the complex and not fully understood pathogenesis of endometriosis.
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Abstract
Endometriosis is a common gynecological condition with a pathogenesis that is multifactorial and not well understood. The aim of this review was to explore the multiplicity of epigenetic changes in the field of this disease.
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References (32)
- Aberrant expression of regulators of cell-fate found in eutopic endometrium is found in matched ectopic endometrium among women and in a baboon model of endometriosis via openalex
- Aberrant methylation at HOXA10 may be responsible for its aberrant expression in the endometrium of patients with endometriosis via openalex
- Coordination of AUF1 and miR-148a destabilizes DNA methyltransferase 1 mRNA under hypoxia in endometriosis via openalex
- Endometriosis: pathogenesis and treatment via openalex
- Endometriotic cells are resistant to interferon-γ-induced cell growth inhibition and apoptosis: a possible mechanism involved in the pathogenesis of endometriosis via openalex
- Epigenetic regulation of the pathological process in endometriosis via openalex
- Evolving Spectrum: via openalex
- Genome-Wide DNA Methylation Analysis Predicts an Epigenetic Switch for GATA Factor Expression in Endometriosis via openalex
- Genome-Wide DNA Methylation Profiling in Cultured Eutopic and Ectopic Endometrial Stromal Cells via openalex
- Hypoxia-Induced MicroRNA-20a Expression Increases ERK Phosphorylation and Angiogenic Gene Expression in Endometriotic Stromal Cells via openalex
- Increased Nuclear Expression of Nuclear Factor Kappa‐B p65 Subunit in the Eutopic Endometrium and Ovarian Endometrioma of Women with Advanced Stage Endometriosis via openalex
- Pathogenesis of endometriosis: The role of defective ‘immunosurveillance’ via openalex
- Polymorphisms in the promoter regions of FAS and FASL genes as candidate genetic factors conferring susceptibility to endometriosis via openalex
- Prolonged stimulation with tumor necrosis factor-α induced partial methylation at PR-B promoter in immortalized epithelial-like endometriotic cells via openalex
- Promoter Hypermethylation of Progesterone Receptor Isoform B (PR-B) in Endometriosis via openalex
- Regulation of Steroidogenic Acute Regulatory Protein Expression and Progesterone Production in Endometriotic Stromal Cells via openalex
- Selective inhibition of prostaglandin E2 receptors EP2 and EP4 modulates DNA methylation and histone modification machinery proteins in human endometriotic cells via openalex
- Theories on the Pathogenesis of Endometriosis via openalex
- Trichostatin A, a Histone Deacetylase Inhibitor, Attenuates Invasiveness and Reactivates E-Cadherin Expression in Immortalized Endometriotic Cells via openalex
- W4392145873 via openalex
- W1976214975 via openalex
- W2009551417 via openalex
- W2030052454 via openalex
- W2044789514 via openalex
- W2066608271 via openalex
- W2074326406 via openalex
- W2114673109 via openalex
- W2130222408 via openalex
- W2141826210 via openalex
- W2155651466 via openalex
- W2163443592 via openalex
- W2343955274 via openalex
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- last seen: 2026-06-10T17:14:06.276822+00:00
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