Deregulation of LOXL1 and HTRA1 Gene Expression in Endometriosis
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This study analyzed gene expression in endometriosis lesions and found LOXL1 and HTRA1 were upregulated, suggesting their involvement in disease pathology.
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References (70)
- Abnormal regulation of chemokine TECK and its receptor CCR9 in the endometriotic milieu is involved in pathogenesis of endometriosis by way of enhancing invasiveness of endometrial stromal cells via openalex
- Angiongenesis: a new theory for endometriosis via openalex
- Apoptosis in normal ovaries of women with and without endometriosis via openalex
- Aromatase: a key molecule in the pathophysiology of endometriosis and a therapeutic target via openalex
- Detection of DNA copy number changes in human endometriosis by comparative genomic hybridization via openalex
- Detection of DNA copy number changes in human endometriosis by comparative genomic hybridization via openalex
- Differentially expressed genes in human endometrial endothelial cells derived from eutopic endometrium of patients with endometriosis compared with those from patients without endometriosis via openalex
- Endometrial Anomalies in Women with Endometriosis via openalex
- Endometriosis: abnormal endometrium and dysfunctional immune response via openalex
- Endometriosis: Current thinking via openalex
- Endometriosis-Specific Genes Identified by Real-Time Reverse Transcription-Polymerase Chain Reaction Expression Profiling of Endometriosis Versus Autologous Uterine Endometrium via openalex
- Expression Profiling of Endometrium from Women with Endometriosis Reveals Candidate Genes for Disease-Based Implantation Failure and Infertility via openalex
- Gelatinase A (MM-2), gelatinase B (MMP-9) and their inhibitors (TIMP 1, TIMP-2) in serum of women with endometriosis: Significant correlation between MMP-2, MMP-9 and their inhibitors without difference in levels of matrix metalloproteinases and tissue inhibitors of metalloproteinases in relation to the severity of endometriosis via openalex
- Gene Expression Profiling of the Rat Endometriosis Model via openalex
- Identification of molecular markers for endometriosis in blood lymphocytes by using deoxyribonucleic acid microarrays via openalex
- Increased peritoneal and endometrial gene expression of biologically relevant cytokines and growth factors during the menstrual phase in women with endometriosis via openalex
- Localization of laminin, fibronectin, E-cadherin, and integrins in endometrium and endometriosis via openalex
- Menstrual effluent in endometriosis shows no difference in volume, VEGF-A, MMP2 and MMP9 or sFLT via openalex
- Molecular mechanisms and biological plausibility underlying the malignant transformation of endometriosis: a critical analysis via openalex
- Regulation of endometrial stromal cell matrix metalloproteinase activity and invasiveness by interleukin-8 via openalex
- Relationship between apoptosis and the number of macrophages in eutopic endometrium from women with and without endometriosis via openalex
- Revised American Society for Reproductive Medicine classification of endometriosis: 1996 via openalex
- Secretion of matrix metalloproteinases by human endometrial cells <i>in vitro</i> via openalex
- Serial analysis of gene expression reveals differential expression between endometriosis and normal endometrium. Possible roles for AXL and SHC1 in the pathogenesis of endometriosis via openalex
- Serum and endometrial markers via openalex
- Serum and peritoneal fluid levels of IGF I and II and insulinlike growth binding protein-3 in endometriosis. via openalex
- Stromal cells of endometriosis fail to produce paracrine factors that induce epithelial 17β-hydroxysteroid dehydrogenase type 2 gene and its transcriptional regulator Sp1: a mechanism for defective estradiol metabolism via openalex
- Symptom distribution among infertile and fertile endometriosis cases with different stages and localisations via openalex
- Urokinase‐Type Plasminogen Activator and Insulin‐Like Growth Factor‐Binding Protein 3 mRNA Expression in Endometriotic Lesions and Eutopic Endometrium via openalex
- Was Sampson wrong? via openalex
- Whole genome deoxyribonucleic acid microarray analysis of gene expression in ectopic versus eutopic endometrium via openalex
- W2139397115 via openalex
- W2141026022 via openalex
- W2150739315 via openalex
- W2163889800 via openalex
- W2164002837 via openalex
- W2168734953 via openalex
- W2183821739 via openalex
- W4211081176 via openalex
- W2092177641 via openalex
- W1966051337 via openalex
- W1973925742 via openalex
- W1999612286 via openalex
- W2001454734 via openalex
- W2006679443 via openalex
- W2007608938 via openalex
- W2018144542 via openalex
- W2019002861 via openalex
- W2035503420 via openalex
- W2037054089 via openalex
- W2045487737 via openalex
- W2049642836 via openalex
- W2052873840 via openalex
- W2054680063 via openalex
- W2059586092 via openalex
- W2065610761 via openalex
- W2068525568 via openalex
- W2085574219 via openalex
- W2091609235 via openalex
- W1499476232 via openalex
- W2092843143 via openalex
- W2093865824 via openalex
- W2094102358 via openalex
- W2100044667 via openalex
- W2102562347 via openalex
- W2107277218 via openalex
- W2117612500 via openalex
- W2127260487 via openalex
- W2127671773 via openalex
- W2129520134 via openalex
Cited by (7)
- Screening and identification of key biomarkers associated with endometriosis using bioinformatics and next generation sequencing data analysis 2024
- Screening and identification of key biomarkers associated with endometriosis using bioinformatics and next-generation sequencing data analysis 2024
- Upregulation of HTRA1 mediated by the lncRNA NEAT1/miR-141-3p axis contributes to endometriosis development through activating NLRP3 inflammasome-mediated pyroptotic cell death and cellular inflammation 2023
- The Apoptotic, Angiogenic and Cell Proliferation Genes CD63, S100A6 e GNB2L1 are Altered in Patients with Endometriosis 2018
- Dysregulation of Lysyl Oxidase Expression in Lesions and Endometrium of Women With Endometriosis 2015
- RHOC: A Key Gene for Endometriosis 2013
- TAGLN expression is deregulated in endometriosis and may be involved in cell invasion, migration, and differentiation 2011
Source provenance
- europepmc
- last seen: 2026-06-11T06:19:48.454388+00:00
- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00
- pubmed
- last seen: 2026-05-13T22:17:00.782903+00:00
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