TAGLN expression is deregulated in endometriosis and may be involved in cell invasion, migration, and differentiation

Dos Santos Hidalgo G, Gabriela dos Santos Hidalgo, Meola J, Juliana Meola, Rosa E Silva JC, Júlio César Rosa e Silva, Cláudia Cristina Paro de Paz, Paro de Paz CC, Ferriani RA, Rui Alberto Ferriani
Fertility and sterility · 2011 · vol. 96(3) , pp. 700–703 · doi:10.1016/j.fertnstert.2011.06.052 · PMID:21763649 · W1970401017
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⚙ AI-generated summary by claude@2026-06, 2026-06-07 ⓘ

This study investigated the role of TAGLN in endometriosis, finding its expression is deregulated and may influence key cellular processes like invasion, migration, and differentiation.

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Abstract

We found an increased expression of the TAGLN gene in endometriotic lesions compared with the eutopic endometrium of the same patients by real-time polymerase chain reaction. It is possible that this deregulation contributes to the development and maintenance of endometriosis by being involved in the pathways of organization of cytoskeletal architecture.

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Condition tags

endometriosis

MeSH descriptors

Cell Movement Endometriosis Endometriosis Endometrium Microfilament Proteins Muscle Proteins Adult Cell Differentiation Cell Differentiation Cell Movement Endometriosis Endometrium Female Gene Expression Regulation Gene Expression Regulation Humans Microfilament Proteins Middle Aged Muscle Proteins Ovarian Diseases

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (36)

Cited by (7)

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