Role of Th1, Th2, Th17, and regulatory T cells in endometriosis
This study investigated the involvement of Th1, Th2, Th17, and regulatory T cells in the pathogenesis of endometriosis by examining their populations and cytokine profiles in affected tissues.
One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works
My notes (saved in your browser only)
Condition tags
Citation neighborhood
Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.
References (100)
- 17β-Estradiol and Lipopolysaccharide Additively Promote Pelvic Inflammation and Growth of Endometriosis via openalex
- 1-Methyl-tryptophan attenuates regulatory T cells differentiation due to the inhibition of estrogen-IDO1-MRC2 axis in endometriosis via openalex
- Association between FOXP3+ regulatory T-cells and occurrence of peritoneal lesions in women with ovarian endometrioma and dermoid cysts via openalex
- Bacterial contamination hypothesis: a new concept in endometriosis via openalex
- Changes in the T-helper cytokine profile and in lymphocyte activation at the systemic and local levels in women with endometriosis via openalex
- Differential Levels of Regulatory T Cells and T-Helper-17 Cells in Women With Early and Advanced Endometriosis via openalex
- Early Endometriosis in Females Is Directed by Immune-Mediated Estrogen Receptor α and IL-6 Cross-Talk via openalex
- Endometriosis: Interaction of Immune and Endocrine Systems via openalex
- Escherichia coli contamination of menstrual blood and effect of bacterial endotoxin on endometriosis via openalex
- Exacerbation of Endometriosis Due To Regulatory T-Cell Dysfunction via openalex
- Higher activity by opaque endometriotic lesions than nonopaque lesions via openalex
- IL-17A Contributes to the Pathogenesis of Endometriosis by Triggering Proinflammatory Cytokines and Angiogenic Growth Factors via openalex
- Immunological Aspects of Endometriosis: An Update via openalex
- Immunological aspects of endometriosis: a review. via openalex
- Increased percentage of Th17 cells in peritoneal fluid is associated with severity of endometriosis via openalex
- Interleukin-4 and Prostaglandin E2 Synergistically Up-Regulate 3β-Hydroxysteroid Dehydrogenase Type 2 in Endometrioma Stromal Cells via openalex
- Interleukin-4 induces expression of eotaxin in endometriotic stromal cells via openalex
- Interleukin-4 Stimulates Proliferation of Endometriotic Stromal Cells via openalex
- Interleukin (IL)-17A Stimulates IL-8 Secretion, Cyclooxygensase-2 Expression, and Cell Proliferation of Endometriotic Stromal Cells via openalex
- Intra-uterine microbial colonization and occurrence of endometritis in women with endometriosis† via openalex
- Lipopolysaccharide promotes the development of murine endometriosis‐like lesions via the nuclear factor‐kappa B pathway via openalex
- Lymphocytes in Endometriosis via openalex
- Molecular detection of intrauterine microbial colonization in women with endometriosis via openalex
- Natural Killer Cells: Key Players in Endometriosis via openalex
- Peritoneal endometriosis due to the menstrual dissemination of endometrial tissue into the peritoneal cavity via openalex
- Peritoneal fluid concentrations of interleukin‐17 correlate with the severity of endometriosis and infertility of this disorder via openalex
- Recruitment of CCR6-Expressing Th17 Cells by CCL 20 Secreted from IL-1β-, TNF-α-, and IL-17A-Stimulated Endometriotic Stromal Cells via openalex
- Regulatory T cells and other leukocytes in the pathogenesis of endometriosis via openalex
- Retrograde menstruation in healthy women and in patients with endometriosis. via openalex
- Role of Estrogen Receptor Signaling Required for Endometriosis-Like Lesion Establishment in a Mouse Model via openalex
- Role of Natural Killer Cell Activity in the Pathogenesis of Endometriosis via openalex
- Role of NK cells and HLA-G in endometriosis via openalex
- Role of TGF- s in normal human endometrium and endometriosis via openalex
- Serum Level of IL-10 Is Increased in Patients with Endometriosis, and IL-10 Promotes the Growth of Lesions in a Murine Model via openalex
- Simultaneous Detection and Evaluation of Four Subsets of CD4+ T Lymphocyte in Lesions and Peripheral Blood in Endometriosis via openalex
- The biological role of Treg cells in ectopic endometrium homeostasis. via openalex
- The involvement of T lymphocytes in the pathogenesis of endometriotic tissues overgrowth in women with endometriosis via openalex
- Toll‐like receptor system and endometriosis via openalex
- T regulatory lymphocytes in patients with endometriosis via openalex
- W2072853625 via openalex
- W2077492907 via openalex
- W2060220862 via openalex
- W2057809738 via openalex
- W2085918020 via openalex
- W2054344840 via openalex
- W2048254575 via openalex
- W2097425822 via openalex
- W2100592302 via openalex
- W2039467806 via openalex
- W2104159258 via openalex
- W2105484520 via openalex
- W2105652521 via openalex
- W2110361388 via openalex
- W2114737175 via openalex
- W2036334866 via openalex
- W2123494997 via openalex
- W2124221440 via openalex
- W2127625228 via openalex
- W2129319296 via openalex
- W2130222408 via openalex
- W2131621879 via openalex
- W2133031521 via openalex
- W2133449490 via openalex
- W2136278733 via openalex
- W2137590150 via openalex
- W2138277367 via openalex
- W2139802799 via openalex
- W2141649011 via openalex
- W2149886990 via openalex
- W2149915530 via openalex
- W2020158930 via openalex
- W2152014155 via openalex
- W2152508754 via openalex
- W2019077533 via openalex
- W2155227851 via openalex
- W2155779547 via openalex
- W2156964052 via openalex
- W2157339124 via openalex
- W2157739158 via openalex
- W2158550801 via openalex
- W2162097247 via openalex
- W2012095515 via openalex
- W2010737442 via openalex
- W2007631222 via openalex
- W2004755026 via openalex
- W2224163882 via openalex
- W2256155471 via openalex
- W1984390733 via openalex
- W2271979548 via openalex
- W1977609213 via openalex
- W2505159449 via openalex
- W2548313353 via openalex
- W2559895200 via openalex
- W1968194779 via openalex
- W1835714742 via openalex
- W2753423724 via openalex
- W1814172467 via openalex
- W2899748139 via openalex
- W1483162601 via openalex
- W141728048 via openalex
Source provenance
- openalex
- last seen: 2026-06-04T00:00:01.174412+00:00
- unpaywall
- last seen: 2026-06-02T02:00:03.124865+00:00