Molecular mechanisms contributing to the pathogenesis of endometriosis
This paper reviews the molecular mechanisms, including altered gene expression, immune dysfunction, and steroid hormone signaling, that contribute to the development and progression of endometriosis.
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References (57)
- Angiogenesis and Macrophage Activation in Endometriosis via openalex
- Angiogenic Factors in Endometriosis via openalex
- Aromatase as a Therapeutic Target in Endometriosis via openalex
- Basal and stimulated secretion of cytokines by peritoneal macrophages in women with endometriosis via openalex
- Cell proliferation is increased in the endometrium of women with endometriosis via openalex
- Detection of chromosomal aneuploidy in endometriosis by multi-color fluorescence in situ hybridization (FISH) via openalex
- Endometriosis: a dysfunction and disease of the archimetra via openalex
- Endometriosis associated with the N314D mutation of galactose-1-phosphate uridyl transferase (GALT) via openalex
- Endometriosis in Rhesus Monkeys (Macaca mulatta) Following Chronic Exposure to 2,3,7,8-Tetrachlorodibenzo-p-dioxin via openalex
- Expression of estrogen receptor alpha and beta in peritoneal and ovarian endometriosis via openalex
- Expression pattern of integrin adhesion molecules in endometriosis and human endometrium via openalex
- Extracellular matrix remodelling in the endometrium and its possible relevance to the pathogenesis of endometriosis via openalex
- Heritable aspects of endometriosis via openalex
- HOX gene expression is altered in the endometrium of women with endometriosis via openalex
- Implantation Defects in Infertile Women with Endometriosis via openalex
- Increased activation of pelvic macrophages in infertile women with mild endometriosis via openalex
- Increased chemotactic activity of peritoneal fluid in patients with endometriosis via openalex
- Increased levels of interleukin-6 and interleukin-10 in the peritoneal fluid of patients with endometriosis via openalex
- Increased microvessel density in adenomyosis uteri via openalex
- Insulin-like growth factors (IGFs), IGF-binding proteins (IGFBPs), and IGFBP-3 protease activity in the peritoneal fluid of patients with and without endometriosis via openalex
- Interferon‐Gamma (IFN‐γ) and Interleukin‐6 (IL‐6) in Peritoneal Fluid and Macrophage‐Conditioned Media of Women With Endometriosis via openalex
- Localization in Tissues and Secretion of Eotaxin by Cells from Normal Endometrium and Endometriosis1 via openalex
- Localization of Matrix Metalloproteinase-2 in Uterine Endometrium and Ectopic Implants via openalex
- Macrophage derived growth factors m odulate Fas ligand expression in cultured endometrial stromal cells: a role in endometriosis via openalex
- Macrophages, Oxidation, and Endometriosis via openalex
- Mechanisms of Development of Prolactin-Induced Adenomyosis in Mice via openalex
- Molekulare Aspekte Zur Angiogenese Und Immunologie Der Endometriose via openalex
- Pathophysiology of adenomyosis via openalex
- Peritoneal endometriosis due to the menstrual dissemination of endometrial tissue into the peritoneal cavity via openalex
- Peritoneal fluid concentrations of the cytokine RANTES correlate with the severity of endometriosis via openalex
- Peritoneal fluid volume, estrogen, progesterone, prostaglandin, and epidermal growth factor concentrations in patients with and without endometriosis via openalex
- Pituitary-ovarian dysfunction and endometriosis via openalex
- Possible involvement of arylamine N-acetyltransferase 2, glutathione S-transferases M1 and T1 genes in the development of endometriosis via openalex
- Progesterone Receptor Isoform A But Not B Is Expressed in Endometriosis via openalex
- Retrograde menstruation in healthy women and in patients with endometriosis. via openalex
- Secondary müllerian system and endometriosis via openalex
- The expression of soluble intercellular adhesion molecule-1 in endometriosis via openalex
- Tracing cellular and molecular mechanisms involved in endometriosis via openalex
- Vascular endothelial growth factor and endometriotic angiogenesis via openalex
- W2142285406 via openalex
- W2092843143 via openalex
- W2058692557 via openalex
- W2043410848 via openalex
- W2039144212 via openalex
- W2152026308 via openalex
- W2025393772 via openalex
- W2000000541 via openalex
- W1972563403 via openalex
- W1949259756 via openalex
- W2406042991 via openalex
- W2412739212 via openalex
- W141728048 via openalex
- W2109604562 via openalex
- W2128319359 via openalex
- W2105981541 via openalex
- W2140246703 via openalex
- W2102562347 via openalex
Cited by (6)
- The nervous system and genomics in endometriosis 2018
- Nerve fibre infiltration and expression in peritoneal lesions of endometriosis in a nonhuman primate model of endometriosis 2018
- The role of the oxidative-stress in the endometriosis-related infertility 2009
- Resolving endometriosis-induced pain utilizing a low-glycemic diet 2008
- [Endometriosis. Is it a problem of the immunological signs?]. 2005
- Ovarielle Stimulation und intrauterine Insemination (COH-IUI) 2004
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