Suppression of IL-1β-induced COX-2 expression by trichostatin A (TSA) in human endometrial stromal cells
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Trichostatin A, a histone deacetylase inhibitor, was found to suppress IL-1β-induced COX-2 expression in human endometrial stromal cells.
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Cited by (38)
- The effects of endometrial polyps, leiomyomas, adenomyosis, and endometriosis on endometrial receptivity: phenotype, molecular pathways, and clinical implications 2026
- Aberrant expression of histone deacetylase 8 in endometriosis and its potential as a therapeutic target 2023
- Epigenetic Dysregulation in Endometriosis: Implications for Pathophysiology and Therapeutics 2023
- Corroborating evidence for aberrant expression of histone deacetylase 8 in endometriosis 2023
- Medical treatments of endometriosis: a review 2022
- Cracking the enigma of adenomyosis: an update on its pathogenesis and pathophysiology 2022
- Perioperative Suppression of Schwann Cell Dedifferentiation Reduces the Risk of Adenomyosis Resulting from Endometrial–Myometrial Interface Disruption in Mice 2022
- Deoxyribonucleic acid and chromatin imaging of endometriosis and endometrial carcinoma using atomic force microscopy 2019
- The Pathogenesis of Endometriosis: Molecular and Cell Biology Insights 2019
- Effects of histone methyltransferase inhibition in endometriosis† 2018
- Current and emerging treatment options for endometriosis 2018
- Investigational drugs for the treatment of endometriosis, an update on recent developments 2018
- Epigenetic Drugs in the Treatment of Endometriosis 2018
- The promising role of placental growth factor in association with cyclooxygenase-2 as predictive markers for recurrent ovarian endometriosis 2017
- Tranylcypromine, a lysine-specific demethylase 1 (LSD1) inhibitor, suppresses lesion growth and improves generalized hyperalgesia in mouse with induced endometriosis 2016
- Platelets are an unindicted culprit in the development of endometriosis: clinical and experimental evidence 2015
- An overview of early drug development for endometriosis 2015
- Selective inhibition of prostaglandin E2 receptors EP2 and EP4 modulates DNA methylation and histone modification machinery proteins in human endometriotic cells 2015
- Potential New Drugs for Endometriosis: Experimental Evidence 2014
- Endometriosis 2013
- Valproic Acid and Progestin Inhibit Lesion Growth and Reduce Hyperalgesia in Experimentally Induced Endometriosis in Rats 2012
- Histone deacetylase inhibitors as therapeutics for endometriosis 2012
- The Epigenetics of Endometriosis 2012
- Dysmenorrhea and its severity are associated with increased uterine contractility and overexpression of oxytocin receptor (OTR) in women with symptomatic adenomyosis 2012
- Interleukin-1β induces cyclooxygenase-2 expression and promotes the invasive ability of human mesenchymal stem cells derived from ovarian endometrioma 2011
- Application of the histone deacetylase inhibitors for the treatment of endometriosis: histone modifications as pathogenesis and novel therapeutic target 2011
- Pathogenesis: Epigenetics 2011
- Valproic acid alleviates generalized hyperalgesia in mice with induced adenomyosis 2011
- The emerging role of epigenetics and miRNAs in endometriosis 2011
- Constitutive and Tumor Necrosis Factor-Alpha-Stimulated Activation of Nuclear Factor-KappaB in Immortalized Endometriotic Cells and Their Suppression by Trichostatin A 2010
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- Peroxisome proliferator-activated receptor-gamma and retinoid X receptor agonists synergistically suppress proliferation of immortalized endometrial stromal cells 2008
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- Trichostatin A, a Histone Deacetylase Inhibitor, Attenuates Invasiveness and Reactivates E-Cadherin Expression in Immortalized Endometriotic Cells 2007
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- pubmed
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