Interleukin-6 and other soluble factors in peritoneal fluid and endometriomas and their relation to pain and aromatase expression

Irene Velasco; Pedro Acién; Adolfo Campos; María Isabel Acién; Maribel Acién; Eva Ruiz-Maciá

doi:10.1016/j.jri.2009.11.004

Interleukin-6 and other soluble factors in peritoneal fluid and endometriomas and their relation to pain and aromatase expression

Irene Velasco, Pedro Acién, Adolfo Campos, María Isabel Acién, Maribel Acién, Eva Ruiz-Maciá

Journal of reproductive immunology · 2010 · vol. 84(2) , pp. 199–205 · doi:10.1016/j.jri.2009.11.004 · PMID:20074813 · W2030699343

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This study investigated the presence of interleukin-6 and other soluble factors in peritoneal fluid and endometriomas, examining their association with pain and aromatase expression.

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mesh:D004412mesh:D004715mesh:D017699

MeSH descriptors

Aromatase Endometriosis Interleukin-6 Pelvic Pain Adult Aromatase Aromatase Ascitic Fluid Ascitic Fluid CA-125 Antigen CA-125 Antigen Dysmenorrhea Endometriosis Endometriosis Endometriosis Female Follow-Up Studies Humans Interleukin-6 Interleukin-6

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[1] Absence of aromatase protein and mRNA expression in endometriosis via openalex

[2] Altered gene expression and secretion of interleukin-6 in stromal cells derived from endometriotic tissues via openalex

[3] Aromatase expression in endometriosis. via openalex

[4] Aromatase expression in endometriotic tissues and cell cultures of patients with endometriosis via openalex

[5] Aromatase expression in endometriotic tissues and its relationship to clinical and analytical findings via openalex

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[7] Changes in cytokine levels of patients with ovarian endometriosis after treatment with gonadotropin-releasing hormone analogue, ultrasound-guided drainage, and intracystic recombinant interleukin-2 via openalex

[8] Characterization of Lymphocyte Subpopulations and T Cell Activation In Endometriosis via openalex

[9] Decreased levels of the potent regulator of monocyte/macrophage activation, interleukin-13, in the peritoneal fluid of patients with endometriosis via openalex

[10] Deficient 17β-Hydroxysteroid Dehydrogenase Type 2 Expression in Endometriosis: Failure to Metabolize 17β-Estradiol<sup>1</sup> via openalex

[11] Different concentrations of interleukins in the peritoneal fluid of women with endometriosis: relationships with lymphocyte subsets via openalex

[12] Ectopic endometrial cells express high concentrations of interleukin (IL)-8 in vivo regardless of the menstrual cycle phase and respond to oestradiol by up-regulating IL-1-induced IL-8 expression in vitro via openalex

[13] GnRH Analogues, Transvaginal Ultrasound-Guided Drainage and Intracystic Injection of Recombinant Interleukin-2 in the Treatment of Endometriosis via openalex

[14] High concentrations of the CA-125, CA 19-9 and CA 15-3 in the peritoneal fluid between patients with and without endometriosis via openalex

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[18] Interleukin-8 Induces Proliferation of Endometrial Stromal Cells: a Potential Autocrine Growth Factor1 via openalex

[19] Interleukin (IL)-17A Stimulates IL-8 Secretion, Cyclooxygensase-2 Expression, and Cell Proliferation of Endometriotic Stromal Cells via openalex

[20] Intraepithelial leukocytes in endometriosis and adenomyosis: comparison of eutopic and ectopic endometrium with normal endometrium via openalex

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[22] Peritoneal fluid concentrations of interleukin‐17 correlate with the severity of endometriosis and infertility of this disorder via openalex

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[25] Prostaglandin E2 Stimulates Aromatase Expression in Endometriosis-Derived Stromal Cells via openalex

[26] Prostaglandin E<sub>2</sub>Stimulates Aromatase Expression in Endometriosis-Derived Stromal Cells<sup>1</sup> via openalex

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