Animal model of uterine adenomyosis: Is prolactin a potent inducer of adenomyosis in mice?

Takao Mori, T Mori, Tippawan Singtripop, T Singtripop, Seiichiro Kawashima, S Kawashima
American Journal of Obstetrics and Gynecology · 1991 · vol. 165(1) , pp. 232–234 · doi:10.1016/0002-9378(91)90258-s · PMID:1853904 · W1967036278
review OA: closed CC0 ⤵ 40 in-corpus citations
Limited metadata. Only one source feed has indexed this record so far — no abstract, full text, or open-access copy is available through Endo Lab. The publisher's page (linked below) is the canonical location for the actual content. If you have institutional access, use "Find at my library".
View at publisher → View on OpenAlex View on PubMed
⚙ AI-generated summary by claude@2026-06, 2026-06-06 ⓘ

This study investigates whether prolactin is a potent inducer of uterine adenomyosis in a mouse model.

One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works

My notes (saved in your browser only)

Condition tags

endometriosisadenomyosis

MeSH descriptors

Endometriosis Prolactin Animals Disease Models, Animal Endometriosis Endometriosis Endometriosis Mice Microscopy, Electron

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (54)

Cited by (40)

Source provenance

europepmc
last seen: 2026-06-13T06:22:48.782012+00:00
openalex
last seen: 2026-06-10T17:14:06.276822+00:00
pubmed
last seen: 2026-05-13T22:12:00.397535+00:00
License: CC0 · commercial use OK