Estrogen Receptor β Modulates Apoptosis Complexes and the Inflammasome to Drive the Pathogenesis of Endometriosis
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Estrogen receptor beta modulates apoptosis complexes and the inflammasome, contributing to the development of endometriosis.
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- The Microbiota–Endometriosis Axis: An Immune–Endocrine Integration Model and Emerging Therapeutic Targets 2026
- Emerging signal transduction pathways and the role of bacterial extracellular vesicles linked to the pathogenesis of endometriosis 2026
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- Electroacupuncture Ameliorates Endometriosis-Associated Ovarian Dysfunction by Activating Nrf2 Pathway to Upregulate GPX4 Function and Inhibiting Ferroptosis 2025
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- Endometriosis and Endometriosis-Associated Tumors 2025
- Identification of Adeno-Associate Virus (AAV) Serotype for Endometriosis Therapy and Effect of AAV-Mediated RNAi Delivery on Gene Expression and Cell Proliferation in In Vitro Endometrial Cell Culture 2025
- Endometriosis as an immune-mediated disease: pathogenetic mechanisms and therapeutic strategies 2025
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- Role of HLA-DRA-CREB3L4 regulatory axis in the pathogenesis of ovarian endometriosis: Inhibition of CREB3L4 expression by HLA-DRA increases the risk of disease 2025
- Dissecting the causal effects of interleukin receptor-related factors and the risk of developing endometriosis: a two-sample Mendelian randomization study 2025
- Endometriosis: pathophysiology and the potential role of diet 2025
- TGF-β/snail-mediated epithelial-to-mesenchymal transition disrupts estradiol metabolism through suppressing the HSD17B2 expression in endometriotic epithelial cells 2025
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- Additional file 1 of Gene expression profiles separate endometriosis lesion subtypes and indicate a sensitivity of endometrioma to estrogen suppressive treatments through elevated ESR2 expression 2024
- Additional file 2 of Causal effects of glycemic traits and endometriosis: a bidirectional and multivariate mendelian randomization study 2024
- Thymol Impacts the Progression of Endometriosis by Disrupting Estrogen Signaling Pathways and Inflammatory Responses 2024
- Endometriosis and Endometriosis-Associated Tumors 2024
- An Indocyanine Green-Based Nanocluster for Imaging Orthodox Endometriosis Lesions with Negative Contrast 2024
- Role of the gut microbiota in the pathogenesis of endometriosis: a review 2024
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- A Primer on Clinical Classification and Pathophysiology of Endometriosis 2024
- The “Road” to Malignant Transformation from Endometriosis to Endometriosis-Associated Ovarian Cancers (EAOCs): An mTOR-Centred Review 2024
- The Association between Endometriosis and Immunological diseases 2024
- Immunologic Aspects of Endometriosis 2024
- Endometriosis: Molecular Pathophysiology and Recent Treatment Strategies—Comprehensive Literature Review 2024
- Estetrol Inhibits Endometriosis Development in an In Vivo Murine Model 2024
- AURKA Enhances the Glycolysis and Development of Ovarian Endometriosis Through ERβ 2024
- NLRP3 concentration, oxidants, and antioxidants in plasma of endometriosis patients undergoing treatment with dienogest 2024
- Endometriosis: Pathogenesis, Diagnosis and Treatment, volume II 2024
- Causal effects of glycemic traits and endometriosis: a bidirectional and multivariate mendelian randomization study 2024
- Identification and exploration of potential N6-methylation related mRNAs in endometriosis 2024
- Meta-analysis and network pharmacology studies of the clinical efficacy of Guizhi Fuling capsules/pills combined with dienogest in treating endometriosis 2024
- Research Progress on the Pathogenesis of Endometriosis 2024
- A Preliminary Investigation of the Roles of Endometrial Cells in Endometriosis Development via In Vitro and In Vivo Analyses 2024
- Additional file 1 of Gene expression profiles separate endometriosis lesion subtypes and indicate a sensitivity of endometrioma to estrogen suppressive treatments through elevated ESR2 expression 2024
- Immune pathway through endometriosis to ovarian cancer 2024
- ERβ-activated LINC01018 promotes endometriosis development by regulating the CDC25C/CDK1/CyclinB1 pathway 2024
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