Genetic Polymorphisms and Molecular Pathogenesis of Endometriosis

Vijayalakshmi Kodati, Q. Has
In: Endometriosis - Basic Concepts and Current Research Trends · 2012 · doi:10.5772/28907 · W1601219650
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This review explores genetic polymorphisms and molecular pathogenesis involved in endometriosis, discussing candidate genes and signaling pathways implicated in the disease.

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This chapter reviews the genetic polymorphisms and molecular mechanisms underlying the pathogenesis of endometriosis, focusing on how specific gene variants contribute to disease susceptibility and progression. It examines various inflammatory pathways, hormone metabolism alterations, and angiogenic factors that are influenced by these genetic differences in affected individuals. The authors highlight significant associations between certain polymorphisms and clinical manifestations, while noting that the complex interplay of environmental and genetic factors makes definitive causal links difficult to establish. This paper is centrally about endometriosis — specifically examining the genetic and molecular basis of its development.

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Abstract

Endometriosis -
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Open access Submitted: 03 March 2011 Published: 09 May 2012 DOI: 10.5772/28907 From the Edited Volume Edited by Koel Chaudhury and Baidyanath Chakravarty Submitted: 03 March 2011 Published: 09 May 2012 © 2012 The Author(s). Licensee IntechOpen. This chapter is distributed under the terms of the Creative Commons Attribution 3.0 License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Discover 2026 Nobel Laureate Francis Halzen’s chapter on IntechOpen >

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endometriosis

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Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

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