The genetics of endometriosis.

The Journal of reproductive medicine · 1998 · vol. 43(3 Suppl) , pp. 263–8 · PMID:9564659 · W299854676
article OA: closed CC0 ⤵ 42 in-corpus citations
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This review examines evidence for endometriosis as a complex genetic trait and describes the OXEGENE study's genome-wide search for susceptibility genes in affected families.

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Abstract

Endometriosis is probably a complex trait, like diabetes, hypertension or asthma, which implies that the disease is caused by an interaction between multiple genes and the environment. This paper reviews the evidence, in both human and nonhuman primates, that supports the theory of a genetic basis to endometriosis. The OXEGENE study, which aims to identify susceptibility genes in endometriosis through linkage analysis, is also described. DNA is being collected from families containing at least two sisters with surgically confirmed revised American Fertility Society stage III and IV disease to carry out a genome-wide search for susceptibility genes. Candidate genes involved in galactose metabolism and dioxin detoxification are also being analyzed. The identification of genes conferring susceptibility to endometriosis may lead to a better understanding of disease etiology and, in time, improved therapeutic strategies and diagnostic methods.

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Condition tags

endometriosis

MeSH descriptors

Endometriosis Genetic Linkage Endometriosis Endometriosis Environmental Pollutants Female Galactose Galactose Galactose Genetic Predisposition to Disease Humans Karyotyping Pedigree

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

Cited by (42)

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