Etiopatogenia da Endometriose

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This dissertation examines the etiopathogenesis of endometriosis, exploring the underlying causes and development of the condition.

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The paper is a dissertation focused on the etiopathogenesis of endometriosis, synthesizing and discussing existing explanations for how the disease develops. It presents a conceptual overview of proposed mechanisms underlying endometriotic lesion formation and progression, drawing on the scientific literature rather than reporting new primary clinical or experimental data. The main limitation is that it is a narrative/overview dissertation, so it does not provide original study-specific results or controlled data comparisons. This paper is centrally about endometriosis — it focuses on etiopathogenesis mechanisms for the disease.

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Please use this identifier to cite or link to this item: https://hdl.handle.net/10216/71754 | Author(s): | Cristina Maria Campos Bragança | | Title: | Etiopatogenia da Endometriose | | Issue Date: | 2013-07-19 | | Subject: | Ciências da saúde Health sciences | | Scientific areas: | Ciências médicas e da saúde::Ciências da saúde Medical and Health sciences::Health sciences | | DOI: | 10.34626/2fxe-we94 | | URI: | https://hdl.handle.net/10216/71754 | | Document Type: | Dissertação | | Rights: | openAccess | | License: | https://creativecommons.org/licenses/by-nc/4.0/ | | Appears in Collections: | ICBAS - Dissertação | Files in This Item: | File | Description | Size | Format | | |---|---|---|---|---| | 30664.pdf | Etiopatogenia da Endometriose | 293.76 kB | Adobe PDF | View/Open | This item is licensed under a Creative Commons License

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Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

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last seen: 2026-06-10T17:14:06.276822+00:00
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