miR-202-3p overexpression attenuates endometriosis-like lesions by modulating YAP-dependent transcription of S100A6 in murine models
article
OA: closed
CC0
⤵ 4 in-corpus citations
Limited metadata. Only one source feed has indexed
this record so far — no abstract, full text, or open-access copy is
available through Endo Lab. The
publisher's page (linked below)
is the canonical location for the actual content. If you have institutional
access, use "Find at my library".
AI-generated summary
Overexpression of miR-202-3p reduced endometriosis-like lesions in mice by suppressing YAP-dependent transcription of S100A6.
One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works
My notes (saved in your browser only)
Condition tags
MeSH descriptors
Citation neighborhood
Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.
References (44)
- Aberrant activation of signal transducer and activator of transcription-3 (STAT3) signaling in endometriosis via openalex
- An Overview of Treatments for Endometriosis via openalex
- A research on the protein expression of p53, p16, and MDM2 in endometriosis via openalex
- Differentially-Expressed miRNAs in Ectopic Stromal Cells Contribute to Endometriosis Development: The Plausible Role of miR-139-5p and miR-375 via openalex
- Effect of S100A6 over‐expression on β‐catenin in endometriosis via openalex
- Endometriosis via openalex
- Endometriosis via openalex
- Endometriosis via openalex
- Endometriosis: pathogenesis and treatment via openalex
- Enhanced miR-210 expression promotes the pathogenesis of endometriosis through activation of signal transducer and activator of transcription 3 via openalex
- Estradiol promotes cells invasion by activating β-catenin signaling pathway in endometriosis via openalex
- Estrogen Receptor β Modulates Apoptosis Complexes and the Inflammasome to Drive the Pathogenesis of Endometriosis via openalex
- Evaluation of the potential therapeutic effects of a double-stranded RNA mimic complexed with polycations in an experimental mouse model of endometriosis via openalex
- Increased expression of YAP is associated with decreased cell autophagy in the eutopic endometrial stromal cells of endometriosis via openalex
- Lentiviral vector-mediated down-regulation of Notch1 in endometrial stem cells results in proliferation and migration in endometriosis via openalex
- MicroRNA-2861 targets STAT3 and MMP2 to regulate the proliferation and apoptosis of ectopic endometrial cells in endometriosis via openalex
- MicroRNA expression profile in endometriosis: its relation to angiogenesis and fibrinolytic factors via openalex
- miRNAs Regulation and Its Role as Biomarkers in Endometriosis via openalex
- Plasma miRNAs as biomarkers for endometriosis via openalex
- Possible involvement of signal transducer and activator of transcription-3 in cell–cell interactions of peritoneal macrophages and endometrial stromal cells in human endometriosis via openalex
- Targeting hypoxia‐mediated YAP1 nuclear translocation ameliorates pathogenesis of endometriosis without compromising maternal fertility via openalex
- The Apoptotic, Angiogenic and Cell Proliferation Genes CD63, S100A6 e GNB2L1 are Altered in Patients with Endometriosis via openalex
- The Origin and Pathogenesis of Endometriosis via openalex
- Use of a Mouse Model of Experimentally Induced Endometriosis to Evaluate and Compare the Effects of Bisphenol A and Bisphenol AF Exposure via openalex
- W6774009233 via openalex
- W2018621705 via openalex
- W2099142804 via openalex
- W2105137499 via openalex
- W2111343488 via openalex
- W2151934245 via openalex
- W2336394695 via openalex
- W2406860897 via openalex
- W2568724647 via openalex
- W2602581116 via openalex
- W2611994759 via openalex
- W2740493453 via openalex
- W2793986555 via openalex
- W2799524602 via openalex
- W2810127666 via openalex
- W2884435515 via openalex
- W2893423916 via openalex
- W2896950141 via openalex
- W2914562289 via openalex
- W2945562893 via openalex
Cited by (4)
- ATG8 inhibited endometriosis formation by regulating Treg cells differentiation via integrin α4β1 and Talin-1 interaction 2023
- miR-206 Targets MALAT1 to Suppress Cell Progression of Ectopic Endometrial Stromal Cells in Endometriosis 2022
- MiR-518c-3p alleviates endometriosis by inhibiting ectopic endometrial migration and epithelial–mesenchymal transition via targeting ZNF608 2022
- Knockdown of miR-150-5p reduces hypoxia-induced autophagy and epithelial-mesenchymal transition of endometriotic cells via regulating the PDCD4/NF-κB signaling pathway 2022
Source provenance
- europepmc
- last seen: 2026-06-11T06:19:48.454388+00:00
- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00
- pubmed
- last seen: 2026-05-13T22:21:36.268089+00:00
- unpaywall
- last seen: 2026-06-02T02:00:03.124865+00:00
License: CC0
· commercial use OK