Knockdown of miR-150-5p reduces hypoxia-induced autophagy and epithelial-mesenchymal transition of endometriotic cells via regulating the PDCD4/NF-κB signaling pathway
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This study investigated the role of miR-150-5p in regulating autophagy and epithelial-mesenchymal transition in endometriotic cells under hypoxic conditions, finding that its knockdown reduced these processes by modulating the PDCD4/NF-κB signaling pathway.
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Cited by (3)
- Upregulation of miR-183 inhibits the invasion and migration of endometrial stromal cells in endometriosis patients by downregulating Ezrin 2025
- Mechanism of the effect of Juan-Tong-Yin on endoplasmic reticulum stress-autophagy in endometriosis rats based on protein kinase R-like endoplasmic reticulum kinase/eukaryotic cell initiation factor 2α pathway 2025
- Research advances in endometriosis-related signaling pathways: A review 2023
Source provenance
- europepmc
- last seen: 2026-06-04T01:30:01.192114+00:00
- openalex
- last seen: 2026-06-04T00:00:01.174412+00:00
- pubmed
- last seen: 2026-06-02T00:34:37.617781+00:00
- unpaywall
- last seen: 2026-06-02T02:00:03.124865+00:00
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