Reduction of estrogen production by interleukin-6 in a human granulosa tumor cell line may have implications for endometriosis-associated infertility
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Interleukin-6 reduced estrogen production in a human granulosa tumor cell line, suggesting a potential mechanism contributing to endometriosis-associated infertility.
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References (33)
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Cited by (17)
- Research advances in endometriosis-related signaling pathways: A review 2023
- Transcriptomic analysis of granulosa cells in patients with endometriosis-related infertility: identification of potential molecular mechanisms 2023
- Novel therapeutic strategy: antiinflammatory reagents 2021
- Pharmaceuticals targeting signaling pathways of endometriosis as potential new medical treatment: A review 2021
- Laparoscopic Surgery Combined with GnRH Agonist in Endometriosis 2019
- Investigating the impact of local inflammation on granulosa cells and follicular development in women with ovarian endometriosis 2019
- Hypoxia‐inhibited DUSP2 expression promotes IL‐6/STAT3 signaling in endometriosis 2017
- Cytokins as the markers for non-invasive diagnosis of pelvic endometriosis 2015
- The Impact of Endometriosis on Fertility 2015
- Anatomy, Development, Histology, and Normal Function of the Ovary 2014
- Inflammation and Cytokines in Endometriosis 2014
- TGF- 1 induces proteinase-activated receptor 2 (PAR2) expression in endometriotic stromal cells and stimulates PAR2 activation-induced secretion of IL-6 2011
- Laparoscopic surgery for endometriosis-associated infertility: a pathophysiologic approach 2010
- 15-Epi-lipoxin A4 inhibits the progression of endometriosis in a murine model 2009
- Interleukin-10 attenuates TNF-α–induced interleukin-6 production in endometriotic stromal cells 2008
- Zur Bedeutung der Endometriose in der Reproduktionsmedizin 2007
- Reduced aromatase activity in granulosa cells of women with endometriosis undergoing assisted reproduction techniques 2006
Source provenance
- europepmc
- last seen: 2026-06-19T06:14:56.452680+00:00
- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00
- pubmed
- last seen: 2026-05-13T22:15:41.664291+00:00
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