Myostatin, follistatin and activin type II receptors are highly expressed in adenomyosis
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This study found that myostatin, follistatin, and activin type II receptors were highly expressed in adenomyosis tissue.
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References (57)
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Cited by (20)
- A holistic and individualised homoeopathic approach to adenomyosis – A case report 2025
- SLC38A2 promotes cell proliferation and invasion by promoting glutamine metabolism in adenomyosis 2024
- Roles of bone morphogenetic proteins in endometrial remodeling during the human menstrual cycle and pregnancy 2023
- Pseudo-invasive Vasculocentric Adenomyosis- A Diagnostic Dilemma 2023
- Contemporary evaluation of women and girls with abnormal uterine bleeding: <scp>FIGO</scp> Systems 1 and 2 2023
- Mechanisms and Pathogenesis of Adenomyosis 2022
- Myostatin: a multifunctional role in human female reproduction and fertility – a short review 2022
- STATE OF CYTOKINE PROFILE IN PATIENTS WITH ADENOMYOSIS 2022
- Insights on Adenomyosis Development 2022
- PIWIL2 is overexpressed in adenomyotic lesions of women with diffuse adenomyosis 2020
- Adenomyosis: Mechanisms and Pathogenesis 2020
- The Effects of Anti-TGF-β1 on Epithelial–Mesenchymal Transition in the Pathogenesis of Adenomyosis 2020
- Role of angiogenesis in adenomyosis-associated abnormal uterine bleeding and subfertility: a systematic review 2019
- Epidemiologic factors associated with endometriosis in East Asia 2019
- Pathogenesis of endometriosis: the genetic/epigenetic theory 2018
- Deep Infiltrating Endometriosis and Endometrial Adenocarcinoma Express High Levels of Myostatin and Its Receptors Messenger RNAs 2017
- Pathogenesis of adenomyosis: an update on molecular mechanisms 2017
- Current and Future Medical Treatment of Adenomyosis 2016
- Decreased Endometrial Expression of Leukemia Inhibitory Factor Receptor Disrupts the STAT3 Signaling in Adenomyosis During the Implantation Window 2016
- Expression of Inflammatory and Neurogenic Mediators in Adenomyosis: A Pathogenetic Role 2016
Source provenance
- europepmc
- last seen: 2026-06-04T01:30:01.192114+00:00
- openalex
- last seen: 2026-06-04T00:00:01.174412+00:00
- pubmed
- last seen: 2026-05-13T22:17:52.213533+00:00
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