Peroxisome proliferating activating receptor gamma–independent attenuation of interleukin 6 and interleukin 8 secretion from primary endometrial stromal cells by thiazolidinediones
Thiazolidinediones were investigated for their ability to reduce interleukin 6 and interleukin 8 secretion from endometrial stromal cells independently of peroxisome proliferating activating receptor gamma.
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Cited by (10)
- The role of peroxisome proliferator-activated receptors in endometriosis 2024
- Endometriosis: A Disease with Few Direct Treatment Options 2022
- Kinase signalling pathways in endometriosis: potential targets for non-hormonal therapeutics 2016
- Progestin suppressed inflammation and cell viability of tumor necrosis factor‐α‐stimulated endometriotic stromal cells 2016
- TNFα-induced IKKβ complex activation influences epithelial, but not stromal cell survival in endometriosis 2016
- Non‐hormonal targets underlying endometriosis: A focus on molecular mechanisms 2015
- Pathogenesis of Endometriosis: Roles of Retinoids and Inflammatory Pathways 2015
- Lipocalin 2 induces the epithelial–mesenchymal transition in stressed endometrial epithelial cells: possible correlation with endometriosis development in a mouse model 2013
- Chemokines in the pathogenesis of endometriosis and infertility 2013
- Regulación del crecimiento del tejido endometrial en respuesta a nuevas alternativas terapéuticas propuestas para tratar la endometriosis 2012
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