The expression of microRNA-451 in human endometriotic lesions is inversely related to that of macrophage migration inhibitory factor (MIF) and regulates MIF expression and modulation of epithelial cell survival

Human Reproduction · 2015 · vol. 30(3) , pp. 642–652 · doi:10.1093/humrep/dev005 · W2130875256
article OA: bronze CC0 ⤵ 33 in-corpus citations
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AI-generated summary by claude@2026-06, 2026-06-07

MicroRNA-451 expression is elevated in endometriotic lesions, where it inversely regulates macrophage migration inhibitory factor and modulates epithelial cell survival.

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Abstract

study question: What is the role of microRNA-451 (miR-451) in human endometriotic tissue? summary answer: miR451 expression was elevated in endometriotic lesion tissue. MiR451 modulated the expression of macrophage migration inhibitory factor and limited cell survival.

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Condition tags

mesh:D004715

MeSH descriptors

Endometriosis Macrophage Migration-Inhibitory Factors MicroRNAs Adult Cells, Cultured Cell Survival Cell Survival Cyclin E Cyclin E Cyclin E Endometriosis Endometriosis Female Gene Expression Regulation Humans Macrophage Migration-Inhibitory Factors Macrophage Migration-Inhibitory Factors MicroRNAs MicroRNAs MicroRNAs

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Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

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