Identification of key modules and candidate genes associated with endometriosis based on transcriptome data via bioinformatics analysis

Weilin Zheng; Dongfang Xiang; Danting Wen; Meicheng Luo; Xuefang Liang; Lixing Cao

doi:10.1016/j.prp.2023.154404

Identification of key modules and candidate genes associated with endometriosis based on transcriptome data via bioinformatics analysis

Weilin Zheng, Dongfang Xiang, Danting Wen, Meicheng Luo, Xuefang Liang, Lixing Cao

Pathology, research and practice · 2023 · vol. 244 , pp. 154404 · doi:10.1016/j.prp.2023.154404 · PMID:36996608 · W4360603678

article OA: closed CC0 ⤵ 7 in-corpus citations

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endometriosis

MeSH descriptors

Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Computational Biology Computational Biology Computational Biology Computational Biology Computational Biology

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References (56)

AP-1 Subunit JUNB Promotes Invasive Phenotypes in Endometriosis via openalex
Association of common variations of the E-cadherin with endometriosis via openalex
Beyond Endometriosis Genome-Wide Association Study: From Genomics to Phenomics to the Patient via openalex
Blood biomarkers for the non-invasive diagnosis of endometriosis via openalex
Cancer-associated mutations in endometriosis: shedding light on the pathogenesis and pathophysiology via openalex
Cellular Changes Consistent With Epithelial–Mesenchymal Transition and Fibroblast-to-Myofibroblast Transdifferentiation in the Progression of Experimental Endometriosis in Baboons via openalex
Differentially expressed gene profiles in the serum before and after the ultrasound-guided ethanol sclerotherapy in patients with ovarian endometriomas via openalex
Dysregulation of the ADAM17/Notch signalling pathways in endometriosis: from oxidative stress to fibrosis via openalex
Endometriosis via openalex
Endometriosis and cancer: what do we know? via openalex
Endometriosis Knowledgebase: a gene-based resource on endometriosis via openalex
Enhanced epithelial to mesenchymal transition (EMT) and upregulated MYC in ectopic lesions contribute independently to endometriosis via openalex
Estrogen Receptor β Modulates Apoptosis Complexes and the Inflammasome to Drive the Pathogenesis of Endometriosis via openalex
Gene Expression Analysis of Endometrium Reveals Progesterone Resistance and Candidate Susceptibility Genes in Women with Endometriosis via openalex
Genes Downregulated in Endometriosis Are Located Near the Known Imprinting Genes via openalex
Genes Linked to Endometriosis by GWAS Are Integral to Cytoskeleton Regulation and Suggests That Mesothelial Barrier Homeostasis Is a Factor in the Pathogenesis of Endometriosis via openalex
Genome-wide genetic analyses highlight mitogen-activated protein kinase (MAPK) signaling in the pathogenesis of endometriosis via openalex
HLA-G is upregulated in advanced endometriosis via openalex
HOXB4 Immunoreactivity in Endometrial Tissues From Women With or Without Endometriosis via openalex
Human Endometrial Fibroblasts Derived from Mesenchymal Progenitors Inherit Progesterone Resistance and Acquire an Inflammatory Phenotype in the Endometrial Niche in Endometriosis1 via openalex
Increased expression of c-fos protein associated with increased matrix metalloproteinase-9 protein expression in the endometrium of endometriotic patients via openalex
Insight into epigenetics of human endometriosis organoids: DNA methylation analysis of HOX genes and their cofactors via openalex
Interplay between Misplaced Müllerian-Derived Stem Cells and Peritoneal Immune Dysregulation in the Pathogenesis of Endometriosis via openalex
Kinase signalling pathways in endometriosis: potential targets for non-hormonal therapeutics via openalex
Medical treatment of endometriosis-related pain via openalex
Molecular Classification of Endometriosis and Disease Stage Using High-Dimensional Genomic Data via openalex
Molecular signature of eutopic endometrium in endometriosis based on the multi-omics integrative synthesis via openalex
New developments in the medical treatment of endometriosis via openalex
Platelets drive smooth muscle metaplasia and fibrogenesis in endometriosis through epithelial–mesenchymal transition and fibroblast-to-myofibroblast transdifferentiation via openalex
Role of NK cells and HLA-G in endometriosis via openalex
Single-cell analysis of menstrual endometrial tissues defines phenotypes associated with endometriosis via openalex
The endometrial immune environment of women with endometriosis via openalex
The role of mitogen‐activated protein kinase signaling pathway in endometriosis via openalex
Towards an understanding of the molecular mechanism of endometriosis: unbalancing epithelial-stromal genetic conflict via openalex
Uncovering Potential Roles of Differentially Expressed Genes, Upstream Regulators, and Canonical Pathways in Endometriosis Using an In Silico Genomics Approach via openalex
World Endometriosis Society consensus on the classification of endometriosis via openalex
W6797865234 via openalex
W2130410032 via openalex
W2171030481 via openalex
W2171875732 via openalex
W2173213419 via openalex
W2297268858 via openalex
W2061096783 via openalex
W82972823 via openalex
W2537623931 via openalex
W2000311445 via openalex
W2766389820 via openalex
W2769365148 via openalex
W3182225233 via openalex
W1966327575 via openalex
W1964847767 via openalex
W4294216483 via openalex
W6608246074 via openalex
W6714839012 via openalex
W6780205309 via openalex
W2163315477 via openalex

Cited by (7)

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License: CC0 · commercial use OK

[1] AP-1 Subunit JUNB Promotes Invasive Phenotypes in Endometriosis via openalex

[2] Association of common variations of the E-cadherin with endometriosis via openalex

[3] Beyond Endometriosis Genome-Wide Association Study: From Genomics to Phenomics to the Patient via openalex

[4] Blood biomarkers for the non-invasive diagnosis of endometriosis via openalex

[5] Cancer-associated mutations in endometriosis: shedding light on the pathogenesis and pathophysiology via openalex

[6] Cellular Changes Consistent With Epithelial–Mesenchymal Transition and Fibroblast-to-Myofibroblast Transdifferentiation in the Progression of Experimental Endometriosis in Baboons via openalex

[7] Differentially expressed gene profiles in the serum before and after the ultrasound-guided ethanol sclerotherapy in patients with ovarian endometriomas via openalex

[8] Dysregulation of the ADAM17/Notch signalling pathways in endometriosis: from oxidative stress to fibrosis via openalex

[9] Endometriosis via openalex

[10] Endometriosis and cancer: what do we know? via openalex

[11] Endometriosis Knowledgebase: a gene-based resource on endometriosis via openalex

[12] Enhanced epithelial to mesenchymal transition (EMT) and upregulated MYC in ectopic lesions contribute independently to endometriosis via openalex

[13] Estrogen Receptor β Modulates Apoptosis Complexes and the Inflammasome to Drive the Pathogenesis of Endometriosis via openalex

[14] Gene Expression Analysis of Endometrium Reveals Progesterone Resistance and Candidate Susceptibility Genes in Women with Endometriosis via openalex

[15] Genes Downregulated in Endometriosis Are Located Near the Known Imprinting Genes via openalex

[16] Genes Linked to Endometriosis by GWAS Are Integral to Cytoskeleton Regulation and Suggests That Mesothelial Barrier Homeostasis Is a Factor in the Pathogenesis of Endometriosis via openalex

[17] Genome-wide genetic analyses highlight mitogen-activated protein kinase (MAPK) signaling in the pathogenesis of endometriosis via openalex

[18] HLA-G is upregulated in advanced endometriosis via openalex

[19] HOXB4 Immunoreactivity in Endometrial Tissues From Women With or Without Endometriosis via openalex

[20] Human Endometrial Fibroblasts Derived from Mesenchymal Progenitors Inherit Progesterone Resistance and Acquire an Inflammatory Phenotype in the Endometrial Niche in Endometriosis1 via openalex

[21] Increased expression of c-fos protein associated with increased matrix metalloproteinase-9 protein expression in the endometrium of endometriotic patients via openalex

[22] Insight into epigenetics of human endometriosis organoids: DNA methylation analysis of HOX genes and their cofactors via openalex

[23] Interplay between Misplaced Müllerian-Derived Stem Cells and Peritoneal Immune Dysregulation in the Pathogenesis of Endometriosis via openalex

[24] Kinase signalling pathways in endometriosis: potential targets for non-hormonal therapeutics via openalex

[25] Medical treatment of endometriosis-related pain via openalex

[26] Molecular Classification of Endometriosis and Disease Stage Using High-Dimensional Genomic Data via openalex

[27] Molecular signature of eutopic endometrium in endometriosis based on the multi-omics integrative synthesis via openalex

[28] New developments in the medical treatment of endometriosis via openalex

[29] Platelets drive smooth muscle metaplasia and fibrogenesis in endometriosis through epithelial–mesenchymal transition and fibroblast-to-myofibroblast transdifferentiation via openalex

[30] Role of NK cells and HLA-G in endometriosis via openalex

[31] Single-cell analysis of menstrual endometrial tissues defines phenotypes associated with endometriosis via openalex

[32] The endometrial immune environment of women with endometriosis via openalex

[33] The role of mitogen‐activated protein kinase signaling pathway in endometriosis via openalex

[34] Towards an understanding of the molecular mechanism of endometriosis: unbalancing epithelial-stromal genetic conflict via openalex

[35] Uncovering Potential Roles of Differentially Expressed Genes, Upstream Regulators, and Canonical Pathways in Endometriosis Using an In Silico Genomics Approach via openalex

[36] World Endometriosis Society consensus on the classification of endometriosis via openalex

[37] W6797865234 via openalex

[38] W2130410032 via openalex

[39] W2171030481 via openalex

[40] W2171875732 via openalex

[41] W2173213419 via openalex

[42] W2297268858 via openalex

[43] W2061096783 via openalex

[44] W82972823 via openalex

[45] W2537623931 via openalex

[46] W2000311445 via openalex

[47] W2766389820 via openalex

[48] W2769365148 via openalex

[49] W3182225233 via openalex

[50] W1966327575 via openalex

[51] W1964847767 via openalex

[52] W4294216483 via openalex

[53] W6608246074 via openalex

[54] W6714839012 via openalex

[55] W6780205309 via openalex

[56] W2163315477 via openalex