Estrogen Stimulates Homing of Endothelial Progenitor Cells to Endometriotic Lesions
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Estrogen administration promoted endothelial progenitor cells to migrate and engraft within endometriotic lesions, suggesting a role for estrogen in lesion neovascularization.
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References (53)
- Angiogenesis and Endometriosis via openalex
- Angiogenesis lymphangiogenesis and neurogenesis in endometriosis via openalex
- Anti-angiogenic treatment strategies for the therapy of endometriosis via openalex
- Blocking of Stromal Cell-Derived Factor-1 Reduces Neoangiogenesis in Human Endometriosis Lesions in a Mouse Model via openalex
- Circulating Endothelial Progenitor Cells Are Up-Regulated in a Mouse Model of Endometriosis via openalex
- Combined blockade of angiotensin II type 1 receptor and activation of peroxisome proliferator-activated receptor- by telmisartan effectively inhibits vascularization and growth of murine endometriosis-like lesions via openalex
- Differential expression of steroidogenic enzymes according to endometriosis type via openalex
- Dual suppression of estrogenic and inflammatory activities for targeting of endometriosis via openalex
- Endometriosis via openalex
- Endometriosis via openalex
- Endometriosis: pathogenesis and treatment via openalex
- High-Resolution Ultrasound Imaging via openalex
- Levels of circulating angiogenic cells are not altered in women with endometriosis via openalex
- Mechanistic and Therapeutic Implications of Angiogenesis in Endometriosis via openalex
- microRNAs expression in endometriosis and their relation to angiogenic factors via openalex
- Resveratrol is a potent inhibitor of vascularization and cell proliferation in experimental endometriosis via openalex
- Size and spatial orientation of uterine tissue transplants on the peritoneum crucially determine the growth and cyst formation of endometriosis-like lesions in mice via openalex
- Vascular development in endometriosis via openalex
- Vascular endothelial growth factor A and C gene expression in endometriosis via openalex
- W2014102827 via openalex
- W2009096648 via openalex
- W2084007114 via openalex
- W2091809756 via openalex
- W2106994385 via openalex
- W2006760151 via openalex
- W2115233843 via openalex
- W1995070910 via openalex
- W2118197078 via openalex
- W2121848641 via openalex
- W2123505593 via openalex
- W1985765304 via openalex
- W2126728673 via openalex
- W2127464720 via openalex
- W2128111376 via openalex
- W1979508193 via openalex
- W2135414744 via openalex
- W2138227261 via openalex
- W2146493861 via openalex
- W2147635498 via openalex
- W2152424657 via openalex
- W1968549922 via openalex
- W2155329562 via openalex
- W2157545113 via openalex
- W2167256358 via openalex
- W1966777109 via openalex
- W2955504961 via openalex
- W1798249409 via openalex
- W2027654614 via openalex
- W6666702564 via openalex
- W2028324779 via openalex
- W2039237411 via openalex
- W2056947224 via openalex
- W2022587663 via openalex
Cited by (19)
- Investigating endometriosis development and endometriosis-related pain over time and in relation to estrogen in a laparoscopic mouse model 2024
- Digestive system deep infiltrating endometriosis: What do we know 2023
- Indole-3-Carbinol Inhibits the Growth of Endometriotic Lesions by Suppression of Microvascular Network Formation 2022
- The ischemic time window of ectopic endometrial tissue crucially determines its ability to develop into endometriotic lesions 2022
- Possible therapeutic effect of royal jelly on endometriotic lesion size, pain sensitivity, and neurotrophic factors in a rat model of endometriosis 2021
- Pain syndrome in adenomyosis. Finding new pathogenesis links and non-hormonal correction opportunities. Literature review 2021
- Assessing Pain Behavioral Responses and Neurotrophic Factors in the Dorsal Root Ganglion, Serum and Peritoneal Fluid in Rat Models of Endometriosis 2021
- Pathophysiologic mechanisms by which adenomyosis predisposes to postpartum haemorrhage and other obstetric complications 2020
- Long noncoding RNAs in endometriosis: Biological functions, expressions, and mechanisms 2020
- Protein kinase CK2 participates in estrogen-mediated endothelial progenitor cell homing to endometriotic lesions through stromal cells in a stromal cell–derived factor-1– CXCR4-dependent manner 2020
- Inhibition of erythropoietin‐producing hepatoma receptor B4 (EphB4) signalling suppresses the vascularisation and growth of endometriotic lesions 2020
- CXCR4 or CXCR7 antagonists treat endometriosis by reducing bone marrow cell trafficking 2020
- Developing Therapies that Prevent Progenitor Cell Migration for Preventing the Development and Further Growth of Endometriotic Lesions- Future Prospects-A Short Communication 2020
- Vascularization of endometrial tissue in abdominal cavity – the most important link in the pathogenesis of endometriosis or its vulnerable spot in terms of treatment? (review of literature) 2019
- Role of angiogenesis in adenomyosis-associated abnormal uterine bleeding and subfertility: a systematic review 2019
- Basic mechanisms of vascularization in endometriosis and their clinical implications 2018
- Oestrogen, progesterone and stem cells: the discordant trio in endometriosis? 2018
- Notch signaling controls sprouting angiogenesis of endometriotic lesions 2017
- Estrogen is essential but not sufficient to induce endometriosis 2017
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- openalex
- last seen: 2026-06-04T00:00:01.174412+00:00
- pubmed
- last seen: 2026-05-13T22:21:00.404924+00:00
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