Dual inhibition of ERK1/2 and AKT pathways is required to suppress the growth and survival of endometriotic cells and lesions
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Dual inhibition of ERK1/2 and AKT pathways effectively suppresses the growth and survival of endometriotic cells and lesions.
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Cited by (13)
- MORPHOLOGICAL FEATURES OF OVARIAN ENDOMETRIOSIS: CHANGES IN OVARIAN TISSUE AND THEIR SIGNIFICANCE FOR REPRODUCTIVE FUNCTION 2026
- Endometrial diseases: a summary of small experimental animal model 2025
- The “Road” to Malignant Transformation from Endometriosis to Endometriosis-Associated Ovarian Cancers (EAOCs): An mTOR-Centred Review 2024
- Synergy between Th1 and Th2 responses during endometriosis: A review of current understanding 2023
- Vanillin prevents the growth of endometriotic lesions through anti-inflammatory and antioxidant pathways in a mouse model 2023
- Sphingosine-1-phosphate receptor 3 is a non-hormonal target to counteract endometriosis-associated fibrosis 2023
- High intensity interval training is superior to moderate intensity continuous training in enhancing the anti-inflammatory and apoptotic effect of pentoxifylline in the rat model of endometriosis 2023
- Targeting the chemerin/CMKLR1 axis by small molecule antagonist α-NETA mitigates endometriosis progression 2022
- Inhibition of Chemerin/CMKLR1 Axis Mitigates the Endometriosis Progression 2022
- 17β-estradiol promotes angiogenesis through non-genomic activation of Smad1 signaling in endometriosis 2021
- Effects of dual inhibition of AKT and ERK1/2 pathways on endometrial pro-inflammatory, hormonal, and epigenetic microenvironment in endometriosis 2021
- The effectiveness of Rutin for prevention of surgical induced endometriosis development in a rat model 2021
- Interleukin-37b inhibits the growth of murine endometriosis-like lesions by regulating proliferation, invasion, angiogenesis and inflammation 2020
Source provenance
- europepmc
- last seen: 2026-06-11T06:19:48.454388+00:00
- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00
- pubmed
- last seen: 2026-05-13T22:19:25.021412+00:00
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