Inhibition of Chemerin/CMKLR1 Axis Mitigates the Endometriosis Progression
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This study investigated the inhibition of the chemerin/CMKLR1 axis and its effect on mitigating endometriosis progression in a mouse model.
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References (59)
- A mouse model of endometriosis that displays vaginal, colon, cutaneous, and bladder sensory comorbidities via openalex
- Bioinformatic analysis reveals the importance of epithelial-mesenchymal transition in the development of endometriosis via openalex
- Dual inhibition of ERK1/2 and AKT pathways is required to suppress the growth and survival of endometriotic cells and lesions via openalex
- Effects of dual inhibition of AKT and ERK1/2 pathways on endometrial pro-inflammatory, hormonal, and epigenetic microenvironment in endometriosis via openalex
- Epithelial–Mesenchymal Transition in Endometriosis—When Does It Happen? via openalex
- Epithelial-to-mesenchymal transition in the development of endometriosis via openalex
- Histone deacetylase HDAC2 silencing prevents endometriosis by activating the HNF4A/ARID1A axis via openalex
- Inhibition of receptor activity–modifying protein 1 suppresses the development of endometriosis and the formation of blood and lymphatic vessels via openalex
- Interplay between Endometriosis and Pregnancy in a Mouse Model via openalex
- Mouse Model of Surgically-induced Endometriosis by Auto-transplantation of Uterine Tissue via openalex
- Mouse Model of Surgically-induced Endometriosis by Auto-transplantation of Uterine Tissue via openalex
- Pharmaceuticals targeting signaling pathways of endometriosis as potential new medical treatment: A review via openalex
- Platelets drive smooth muscle metaplasia and fibrogenesis in endometriosis through epithelial–mesenchymal transition and fibroblast-to-myofibroblast transdifferentiation via openalex
- Rethinking mechanisms, diagnosis and management of endometriosis via openalex
- Role of Estrogen Receptor Signaling Required for Endometriosis-Like Lesion Establishment in a Mouse Model via openalex
- The establishment of a mouse model of deep endometriosis via openalex
- The Origin and Pathogenesis of Endometriosis via openalex
- The role of mesenchymal–epithelial transition in endometrial function via openalex
- W2528136962 via openalex
- W2530458202 via openalex
- W2744378930 via openalex
- W2778410020 via openalex
- W2803033229 via openalex
- W2890467009 via openalex
- W2901068426 via openalex
- W2907385580 via openalex
- W2916992870 via openalex
- W2943893201 via openalex
- W2944501272 via openalex
- W2967335028 via openalex
- W2975383676 via openalex
- W3017301236 via openalex
- W3040264348 via openalex
- W3084449299 via openalex
- W3087309713 via openalex
- W3113320117 via openalex
- W3130950762 via openalex
- W3134885188 via openalex
- W3165294846 via openalex
- W3185464879 via openalex
- W1524779889 via openalex
- W4234160457 via openalex
- W1939810769 via openalex
- W1968846807 via openalex
- W1977282990 via openalex
- W1982368439 via openalex
- W1986082361 via openalex
- W2015466197 via openalex
- W2083441410 via openalex
- W2092983959 via openalex
- W2096588853 via openalex
- W2102800356 via openalex
- W2105942603 via openalex
- W2108932120 via openalex
- W2120307848 via openalex
- W2132767601 via openalex
- W2154589460 via openalex
- W2168015220 via openalex
- W2169173583 via openalex
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