17β-estradiol promotes angiogenesis through non-genomic activation of Smad1 signaling in endometriosis
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17β-estradiol promotes angiogenesis in endometriosis via non-genomic activation of Smad1 signaling, contributing to the disease's development.
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References (41)
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Cited by (5)
- Large Peritoneal Macrophages Promote the Resolution of Inflammation in Injured Endometrium 2025
- Neuroendocrine–Immune Axis in Endometriosis: A Review on How the Nervous System Goes Beyond Pain Perception 2025
- The Scribble-Rac1 signaling axis drives epithelial cell motility and contributes to diffuse adenomyosis pathogenesis 2025
- An Estrogen–NK Cells Regulatory Axis in Endometriosis, Related Infertility, and Miscarriage 2024
- Research advances in endometriosis-related signaling pathways: A review 2023
Source provenance
- europepmc
- last seen: 2026-06-04T01:30:01.192114+00:00
- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00
- pubmed
- last seen: 2026-05-13T22:24:08.918168+00:00
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