The Animal Model of Adenomyosis

In: Uterine Adenomyosis · 2015 · pp. 123–127 · doi:10.1007/978-3-319-13012-5_7 · W2299323489
book-chapter OA: closed CC0 ⤵ 1 in-corpus citation
Limited metadata. Only one source feed has indexed this record so far — no abstract, full text, or open-access copy is available through Endo Lab. The publisher's page (linked below) is the canonical location for the actual content. If you have institutional access, use "Find at my library".
View at publisher → View on OpenAlex
AI-generated summary by claude@2026-06+body, 2026-06-07

Adenomyosis occurs spontaneously in animals and can be induced in laboratory animals, but the functional significance and relevance to humans of existing mouse models remain unclear.

One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works

AI-generated deep summary by claude@2026-06, 2026-06-07

This chapter reviews how spontaneous adenomyosis in multiple animal species has been modeled experimentally, emphasizing that induction depends on species/strain and on the timing of hormonal or endocrine interventions. It highlights neonatal tamoxifen administration and altered prolactin production in mice as the most widely reported models, but notes that the functional significance of adenomyosis in these mice and the relevance of the models to human disease remain unclear. A major limitation acknowledged is the difficulty in translating findings because model formation is highly variable across experimental setups and species. This paper is centrally about endometriosis and/or adenomyosis — specifically, it focuses on animal models of adenomyosis and discusses their relevance to human disease.

Read from the paper's body, not the abstract. Not a substitute for reading the paper. No clinical advice. How this works

My notes (saved in your browser only)

Condition tags

adenomyosis

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (47)

Cited by (1)

Source provenance

openalex
last seen: 2026-06-10T17:14:06.276822+00:00
License: CC0 · commercial use OK