Regulation of progesterone receptor expression in endometriosis, endometrial cancer, and breast cancer by estrogen, polymorphisms, transcription factors, epigenetic alterations, and ubiquitin-proteasome system
This paper explores the regulation of progesterone receptor expression in endometriosis, endometrial cancer, and breast cancer through estrogen, genetic variations, transcription factors, epigenetic changes, and the ubiquitin-proteasome system.
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Cited by (9)
- The Microbiota–Endometriosis Axis: An Immune–Endocrine Integration Model and Emerging Therapeutic Targets 2026
- Biomaterial Tools for Modeling Endometriosis 2026
- Features of treatment of patients with hyperplastic processes in the endometrium in combination with chronic endometritis 2025
- EZH2 promotes endometriosis progression through estrogen receptor and TNFα expression 2025
- Bioorganic Mechanism of Progestins in Endometrial Cancer: Insights from Computational Studies 2025
- Exploration of Diagnostic Deubiquitinating Enzymes in Endometriosis and Its Immune Infiltration 2024
- Commonalities and Disparities between Endometriosis and Chronic Endometritis: Therapeutic Potential of Novel Antibiotic Treatment Strategy against Ectopic Endometrium 2023
- Effects of estradiol, progesterone or cAMP on expression of PGRMC1 and progesterone receptor in a xenograft model of human endometrium and in endometrial cell culture 2023
- Therapeutic effects of Huayu Jiedu formula on endometriosis via downregulating GATA 6 expression 2023
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