Mitochondria-Associated Molecular Mechanisms in Endometriosis - A Mini Review

W Tana
In: Open Access Journal of Gynecology · 2022 · vol. 7(4) , pp. 1–6 · doi:10.23880/oajg-16000247 · W4367336325
Review OA: diamond CC0 ⤵ 1 in-corpus citation
🔓 Open OA copy View on OpenAlex View at publisher
⚙ AI-generated summary by gemini-2.5-flash-lite, 2026-06-06 ⓘ

This review assesses the role of mitochondria in endometriosis, focusing on their regulation of ROS, energy metabolism, and signal transduction in disease development.

One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works

Abstract

Endometriosis is a multifaceted disorder, with a variety of factors and processes at play. Research is currently investigating the influence of genetics, epigenetics, signaling pathways, and oxidative stress in its etiology. It is clear that these elements have an effect on the mitochondria in different cells, which are responsible for regulating ROS production, energy metabolism, and signal transduction in endometriosis. An assessment of mitochondrial-related molecular mechanisms in endometriosis is in progress.

My notes (saved in your browser only)

Condition tags

endometriosis

Citation neighborhood (2-hop)

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. Outer rings show 2-hop neighbours — papers reached through the immediate citers/citees. [ collapse to 1-hop ]

References (30)

Cited by (1)

Source provenance

openalex
last seen: 2026-06-10T17:14:06.276822+00:00
License: CC0 · commercial use OK