Mitochondria-Associated Molecular Mechanisms in Endometriosis - A Mini Review

W Tana
In: Open Access Journal of Gynecology · 2022 · vol. 7(4) , pp. 1–6 · doi:10.23880/oajg-16000247 · W4367336325
review OA: diamond CC0 ⤵ 1 in-corpus citation
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⚙ AI-generated summary by claude@2026-06, 2026-06-06 ⓘ

This review assesses the role of mitochondria in endometriosis, focusing on their regulation of ROS, energy metabolism, and signal transduction in disease development.

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Abstract

Endometriosis is a multifaceted disorder, with a variety of factors and processes at play. Research is currently investigating the influence of genetics, epigenetics, signaling pathways, and oxidative stress in its etiology. It is clear that these elements have an effect on the mitochondria in different cells, which are responsible for regulating ROS production, energy metabolism, and signal transduction in endometriosis. An assessment of mitochondrial-related molecular mechanisms in endometriosis is in progress.

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Condition tags

endometriosis

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

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last seen: 2026-06-10T17:14:06.276822+00:00
License: CC0 · commercial use OK