Mortality risk associated with endometriosis: a systematic review and meta-analysis

meta-analysis OA: closed public-domain-us
View on OpenAlex View on PubMed View at publisher
AI-generated summary by claude@2026-07, 2026-07-18

This meta-analysis found no significant increase in all-cause mortality or mortality from specific diseases among women with endometriosis compared to the general population.

One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works

Abstract

INTRODUCTION: Endometriosis is a chronic, estrogen-dependent gynecological disorder affecting approximately 10% of women worldwide that can cause pelvic pain, lead to infertility, and impair quality of life. Whether the condition elevates risk of mortality remains controversial. This systematic literature review and meta-analysis investigated the long-term survival of women with endometriosis. METHOD: Studies reporting mortality among women with endometriosis were identified through PubMed, EMBASE, Web of Science, Scopus, and OpenGrey in accordance with PRISMA recommendations. Pooled data on all-cause mortality and mortality for specific diseases were meta-analyzed using a random-effects model. Heterogeneity was evaluated using the I2 statistic. RESULTS: Women with endometriosis were not at significantly higher risk than the general population of women for mortality related to gynecological cancer (HR 1.56, 95% CI 0.72-3.39), non-cancerous cardiocerebral disease (HR 0.80, 95% CI 0.53-1.23), non-cancerous respiratory disease (HR 0.89, 95% CI 0.20-4.02), accidents/suicide (HR 1.33, 95% CI 0.99-1.79) or all causes combined (HR 1.00, 95% CI 0.82-1.21). CONCLUSION: Endometriosis does not appear to increase risk of all-cause mortality or mortality due to specific diseases. Future research should focus on how interventions or management influence other long-term outcomes, such as quality of life and mental and reproductive health.

My notes (saved in your browser only)

Condition tags

endometriosisinfertility

MeSH descriptors

Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (35)

Source provenance

europepmc
last seen: 2026-08-01T06:07:04.264727+00:00
openalex
last seen: 2026-08-01T06:00:24.223380+00:00
pubmed
last seen: 2026-08-01T06:02:23.048493+00:00
unpaywall
last seen: 2026-05-15T02:00:00.661756+00:00
License: public-domain-us · commercial use OK · attribution required
Courtesy of the U.S. National Library of Medicine