Effects of Prenatal Environmental Exposures on the Development of Endometriosis in Female Offspring

Ming Wei, Xinyuan Chen, Ye Zhao, Baoli Cao, Wenli Zhao
Reproductive sciences (Thousand Oaks, Calif.) · 2016 · vol. 23(9) , pp. 1129–1138 · doi:10.1177/1933719116630418 · PMID:26905420 · W2465297398
review OA: closed CC0 ⤵ 11 in-corpus citations
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⚙ AI-generated summary by claude@2026-06+body, 2026-06-07 ⓘ

Prenatal exposure to estrogenic substances and environmental toxins may increase the incidence of endometriosis, while cigarette smoke may offer protection.

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⚙ AI-generated deep summary by claude@2026-06, 2026-06-07 ⓘ

This paper is a review analyzing existing studies on how prenatal environmental exposures affect the development of endometriosis in female offspring, drawing on evidence from human epidemiologic studies and animal experiments that examine endocrine disruptors and toxins. The authors report that prenatal exposure to estrogenic substances (e.g., ethinyl estradiol, diethylstilbestrol) and environmental toxins (e.g., TCDD, polychlorinated biphenyls, bisphenol A) may increase the incidence of endometriosis, while cigarette smoke exposure is described as potentially protective, proposed to act via antiestrogenic effects. A key caveat highlighted is that findings across studies have not always been conclusive, leaving the relationships “debatable.” This paper is centrally about endometriosis — it specifically summarizes and analyzes evidence linking prenatal environmental exposures to endometriosis development in female offspring.

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Condition tags

endometriosis

MeSH descriptors

Endometriosis Maternal Exposure Prenatal Exposure Delayed Effects Animals Benzhydryl Compounds Benzhydryl Compounds Bisphenol A Compounds Dioxins Dioxins Endometriosis Endometriosis Ethinyl Estradiol Ethinyl Estradiol Female Humans Maternal Exposure Phenols Phenols Polychlorinated Biphenyls Polychlorinated Biphenyls

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (96)

Cited by (11)

Source provenance

europepmc
last seen: 2026-06-20T06:14:18.781669+00:00
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