Endometriosis and Endosalpingiosis

In: Atlas of Gynaecological Pathology · 1983 · pp. 143–146 · doi:10.1007/978-94-015-7312-2_31 · W234950604
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This paper distinguishes endometriosis and adenomyosis as distinct conditions with different origins despite their frequent co-occurrence in patients.

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This chapter discusses endometriosis and endosalpingiosis within a gynaecological pathology framework, focusing on the conceptual distinction between adenomyosis and endometriosis. It reviews the terminology that groups adenomyosis as “internal endometriosis” and endometriosis as “external endometriosis,” concluding that this terminology lacks valid justification because it obscures that adenomyosis and endometriosis have differing aetiology and pathogenesis even when they can co-occur in the same patient. A major caveat is that the presented argument is conceptual/pathological rather than based on a specific new study with detailed methods or quantitative outcomes. This paper is centrally about endometriosis — it challenges the “internal vs external endometriosis” terminology and emphasizes differences in aetiology and pathogenesis between adenomyosis and endometriosis.

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Abstract

Endometriosis is characterized by the presence of ectopic endometrial tissue in an extra-myometrial site. Attempts have been made to impose a conceptual unity on the two conditions of adenomyosis and endometriosis by referring to the former as ‘internal endometriosis’ and to the latter as ‘external endometriosis’: there is, however, no valid justification for this terminology which blurs the fact that adenomyosis and endometriosis, though not uncommonly occurring together in the same patient, are conditions of differing aetiology and pathogenesis. Preview Unable to display preview. Download preview PDF. Similar content being viewed by others

References

Simpson, J. L., Elias, S., Malinak, L. R. and Buttram, V. C. ( 1980. Heritable aspects of endometriosis. I. Genetic studies. Am. J. Obstet. Gynecol., 137, 327–331 Malinak, L. R., Buttram, V. C., Elias, S. and Simpson, J. L. (1980). Heritable aspects of endometriosis. II. Clinical characteristics of familial endometriosis. Am. J. Obstet. Gynecol„ 137, 332–337 Weed, J. and Arquembourg, P. C. (1980). Endometriosis: can it produce an autoimmune response resulting in infertility? C/in. Obstet. Gynecol., 22, 169 - -186 Czernobilsky, B. (1982). Primary epithelial tumors of the ovary. In Blaustein, A. (ed.). Pathology of the Fema/e Genital Tract. 2nd edn., pp. 511–560. ( New York, Heidelberg, Berlin: Springer-Verlag ) Brooks, J. J. and Wheeler, J. E. (1977). Malignancy arising in extragonadal endometriosis: a case report and summary of the world literature. Cancer, 40, 3065–3073 Mostoufizadeh, M. and Scully, R. E. (1980). Malignant tumors arising in endometriosis. Clin. Obstet. Gynecol., 23, 951–963 Czernobilsky, B. and Morris, W. J. (1979). A histologic study of ovarian endometriosis with emphasis on hyperplastic and atypical changes. Obstet. Gynecol., 53, 318–323 Zinsser, K. R. and Wheeler, J. E. (1982). Endosalpingiosis in the omentum: a study of autopsy and surgical material. Am. J. Surg. Pathol., 6, 109–117 Author information Authors and Affiliations Rights and permissions Copyright information © 1983 H. Fox and C. H. Buckley About this chapter Cite this chapter Fox, H., Buckley, C.H. (1983). Endometriosis and Endosalpingiosis. In: Atlas of Gynaecological Pathology. Current Histopathology, vol 5. Springer, Dordrecht. https://doi.org/10.1007/978-94-015-7312-2_31 Download citation DOI: https://doi.org/10.1007/978-94-015-7312-2_31 Publisher Name: Springer, Dordrecht Print ISBN: 978-94-015-7314-6 Online ISBN: 978-94-015-7312-2 eBook Packages: Springer Book Archive

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Condition tags

endometriosis

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Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (10)

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