Xin Huang

No ORCID on file · 57 papers in corpus · active 2012-2026

Study types

  • preprint 23
  • article 21
  • other 8
  • supplementary-materials 2
  • letter 1

Condition tags

  • endometriosis 55
  • infertility 9
  • dysmenorrhea 3
  • dyspareunia 2
  • chronic_pelvic_pain 1
preprint 2023
·doi:10.1158/1078-0432.22455185.v1

Suppl Fig S3. Antibody-mediated, complement-induced cytotoxicity

preprint 2023
·doi:10.1158/1078-0432.22455182.v1

Suppl Fig S4. Complement and non-target siRNA

preprint 2023
·doi:10.1158/1078-0432.22455182

Suppl Fig S4. Complement and non-target siRNA

preprint 2023
·doi:10.1158/1078-0432.22455179.v1

Suppl Table S1. Differentially expressed genes from different group comparisons

preprint 2023
·doi:10.1158/1078-0432.22455179

Suppl Table S1. Differentially expressed genes from different group comparisons

preprint 2023
·doi:10.1158/1078-0432.22455176

Suppl Table S2. Top five canonical pathways from different group comparisons, identified via Ingenuity Pathway Analyses

preprint 2023
·doi:10.1158/1078-0432.22455176.v1

Suppl Table S2. Top five canonical pathways from different group comparisons, identified via Ingenuity Pathway Analyses

preprint 2023
·doi:10.1158/1078-0432.22455173

Suppl Table S3. Top five networks from different group comparisons, identified via Ingenuity Pathway Analyses

preprint 2023
·doi:10.1158/1078-0432.22455173.v1

Suppl Table S3. Top five networks from different group comparisons, identified via Ingenuity Pathway Analyses

other 2022
Reproductive sciences (Thousand Oaks, Calif.) ·doi:10.1007/s43032-022-00869-3

The prevalence of endometriosis is approximately 10% in women of reproductive age, and 30-50% of women with endometriosis are infertile. Metformin has been reported to inhibit the growth of ectopic lesions in endometriosis. However, its eff…

other 2022
Reproductive sciences (Thousand Oaks, Calif.) ·doi:10.1007/s43032-022-00873-7
article 2022
Frontiers in endocrinology ·doi:10.3389/fendo.2022.813165

We first reported that the Hippo-YAP signaling pathway plays a critical role in the pathogenesis of endometriosis (EMS). Autophagy is also related to the invasion ability of endometrial cells and is involved in the pathogenesis of EMS throu…

article 2022
Journal of Obstetrics and Gynaecology Research ·doi:10.1111/jog.15526

AIM: Progesterone resistance is an epigenetic factor that reduces endometrial receptivity and causes implantation failure in women with endometriosis. In addition, dysregulated miRNAs contribute to the underlying pathogenic mechanisms of en…

preprint 2021
·doi:10.21203/rs.3.rs-145883/v1

Abstract Background: The infertile patients with endometriosis had a higher prevalence of endometrial polyps. The effect of endometrial polyps on pregnancy outcomes in endometriosis-associated infertility patients is unclear. This study ass…

2021
Frontiers in microbiology ·doi:10.3389/fmicb.2021.729744
article 2020
·doi:10.1016/j.fertnstert.2020.09.083
article 2019
Molecular and cellular endocrinology ·doi:10.1016/j.mce.2019.04.012

Basic research has shown that signal pathways play significant roles in the development and progression of endometriosis (EMS). We first reported that the Hippo-YAP (Yes-associated protein) pathway promotes cell proliferation and anti-apopt…

article 2019
·doi:10.1016/j.fertnstert.2019.07.949
article 2019
·doi:10.1210/js.2019-sat-198

Endometriosis (EMS) is a debilitating gynecologic disease characterized by the presence of endometrial glands and stroma outside the uterus. Symptoms caused by this disease have serious implications for women's lives and health. Although it…

other 2018
Reproductive sciences (Thousand Oaks, Calif.) ·doi:10.1177/1933719117734320

This study aimed to study the expression of homeobox (HOX)A11-AS1 ( HOXA11 antisense RNA) long noncoding RNA (lncRNA) and the expression of homeobox A ( HOXA9, HOXA10, HOXA11, and HOXA13) genes in the eutopic (EU) and ectopic (EC) endometri…

article 2018
Molecular medicine reports ·doi:10.3892/mmr.2018.9039

The focus of the current study was a G protein‑coupled estrogen receptor (GPER)/microRNA (miR)‑148a/human leukocyte antigen‑G (HLA‑G) signaling pathway in ovarian endometriosis. Reverse transcription‑quantitative polymerase chain reaction w…

article 2015
·doi:10.1016/j.fertnstert.2015.07.395
article 2015
Oncoimmunology ·doi:10.1080/2162402x.2014.1002732

Immune escape is consequential for cancer development. Identifying abnormalities of the immune microenvironment during early carcinogenesis can provide insight into disease pathogenesis and unravel new preventive or therapeutic targets. We …

article 2014
·doi:10.1158/1538-7445.am2014-1653

Abstract Introduction: Endometriosis is a largely benign, chronic inflammatory disease defined by the presence of endometrial-like glands surrounded by stroma. Epidemiologic studies suggest that endometriosis is an independent risk factor f…

article 2014
Clinical cancer research : an official journal of the American Association for Cancer Research ·doi:10.1158/1078-0432.ccr-14-1338

PURPOSE: Mechanisms of immune dysregulation associated with advanced tumors are relatively well understood. Much less is known about the role of immune effectors against cancer precursor lesions. Endometrioid and clear-cell ovarian tumors p…