Study types
- preprint 18
- article 6
Condition tags
- endometriosis 24
Top journals
Frequent coauthors
- Robert P. Edwards 24
- Anda M Vlad 24
- Esther Elishaev 24
- Tianzhou Ma 22
- Swati Suryawanshi 22
- Xin Huang 21
- Raluca A. Budiu 21
- Sung Hwan Kim 21
- Ted Lee 20
- Suketu Mansuria 20
AbstractPurpose: Mechanisms of immune dysregulation associated with advanced tumors are relatively well understood. Much less is known about the role of immune effectors against cancer precursor lesions. Endometrioid and clear-cell ovarian …
AbstractPurpose: Mechanisms of immune dysregulation associated with advanced tumors are relatively well understood. Much less is known about the role of immune effectors against cancer precursor lesions. Endometrioid and clear-cell ovarian …
Supplementary Materials and Methods
Supplementary Materials and Methods
Suppl Fig S1. Venn diagram of DE genes from 3 group comparisons
Suppl Fig S1. Venn diagram of DE genes from 3 group comparisons
Suppl Fig S2. Kras and Pten pathways in relationship to complement gene expression
Suppl Fig S2. Kras and Pten pathways in relationship to complement gene expression
Suppl Fig S3. Antibody-mediated, complement-induced cytotoxicity
Suppl Fig S3. Antibody-mediated, complement-induced cytotoxicity
Suppl Fig S4. Complement and non-target siRNA
Suppl Fig S4. Complement and non-target siRNA
Suppl Table S1. Differentially expressed genes from different group comparisons
Suppl Table S1. Differentially expressed genes from different group comparisons
Suppl Table S2. Top five canonical pathways from different group comparisons, identified via Ingenuity Pathway Analyses
Suppl Table S2. Top five canonical pathways from different group comparisons, identified via Ingenuity Pathway Analyses
Suppl Table S3. Top five networks from different group comparisons, identified via Ingenuity Pathway Analyses
Suppl Table S3. Top five networks from different group comparisons, identified via Ingenuity Pathway Analyses
Abstract Objectives: To investigate changes in estrogen receptor–alpha (ERα) signaling during the progression of endometriosis to endometriosis-associated ovarian cancer (EAOC) as a putative driver of malignant transformation. Methods: We p…
Abstract Introduction: Endometriosis is a largely benign, chronic inflammatory disease defined by the presence of endometrial-like glands surrounded by stroma. Epidemiologic studies suggest that endometriosis is an independent risk factor f…
PURPOSE: Mechanisms of immune dysregulation associated with advanced tumors are relatively well understood. Much less is known about the role of immune effectors against cancer precursor lesions. Endometrioid and clear-cell ovarian tumors p…
Abstract Endometriosis is a largely benign, chronic inflammatory disease that may predispose to some of the ovarian tumors histotypes, collectively called endometriosis-associated ovarian cancers (EAOC). Although it is widely accepted that …