Additional file 6 of Unraveling pathogenesis, biomarkers and potential therapeutic agents for endometriosis associated with disulfidptosis based on bioinformatics analysis, machine learning and experiment validation
This study presents a network analysis associating signature genes with various categories of drugs, including approved, investigational, nutraceutical, and veterinary-approved medications.
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Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. Outer rings show 2-hop neighbours — papers reached through the immediate citers/citees. [ collapse to 1-hop ]
References (92)
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- All-Trans Retinoic Acid Inhibits Vascular Endothelial Growth Factor Expression in a Cell Model of Neutrophil Activation via openalex
- Ameliorative effects of luteolin against endometriosis progression in vitro and in vivo via openalex
- Bioinformatic analysis reveals the importance of epithelial-mesenchymal transition in the development of endometriosis via openalex
- Co-operation between the AKT and ERK signaling pathways may support growth of deep endometriosis in a fibrotic microenvironment in vitro† via openalex
- Endometriosis via openalex
- Endometriosis via openalex
- Endometriosis and ovarian cancer: potential benefits and harms of screening and risk-reducing surgery via openalex
- Enhanced epithelial to mesenchymal transition (EMT) and upregulated MYC in ectopic lesions contribute independently to endometriosis via openalex
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- Retinoic Acid Biosynthesis Is Impaired in Human and Murine Endometriosis1 via openalex
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- last seen: 2026-05-11T08:50:40.763824+00:00