Histone deacetylase inhibitors down-regulate G-protein-coupled estrogen receptor and the GPER-antagonist G-15 inhibits proliferation in endometriotic cells
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Histone deacetylase inhibitors down-regulate G-protein-coupled estrogen receptor, and the GPER antagonist G-15 inhibits endometriotic cell proliferation.
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References (34)
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Cited by (16)
- Drug development for adenomyosis based on pathophysiology 2025
- Regulatory mechanism of GPER in the invasion and migration of ectopic endometrial stromal cells in endometriosis 2023
- Corroborating evidence for aberrant expression of histone deacetylase 8 in endometriosis 2023
- Aberrant expression of histone deacetylase 8 in endometriosis and its potential as a therapeutic target 2023
- The Impact of Histone Modifications in Endometriosis Highlights New Therapeutic Opportunities 2023
- Advances in targeting estrogen synthesis and receptors in patients with endometriosis 2022
- Histone deacetylase 1, Sirtuin 1, and Sirtuin 3 single-nucleotide polymorphisms and the risk of endometriosis in South Indian women 2022
- Spatial and temporal changes in the expression of steroid hormone receptors in mouse model of endometriosis 2020
- Endometriosis 2020
- microRNA and Overcoming the Challenges of Their Use in the Diagnosis of Endometriosis 2020
- Recent insights on the genetics and epigenetics of endometriosis 2016
- Treatment of Endometriosis with Non-Hormonal Agents: Review 2016
- G protein–coupled estrogen receptor 1 agonist G-1 induces cell cycle arrest in the mitotic phase, leading to apoptosis in endometriosis 2015
- Innovations in classical hormonal targets for endometriosis 2015
- Association between DNMT3L polymorphic variants and the risk of endometriosis-associated infertility 2015
- Aberrant histone modification in endometriosis 2014
Source provenance
- europepmc
- last seen: 2026-06-04T01:30:01.192114+00:00
- openalex
- last seen: 2026-06-04T00:00:01.174412+00:00
- pubmed
- last seen: 2026-05-13T22:18:59.468224+00:00
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