Comparison of Buserelin to Danazol Therapy in Endometriosis

Although the etiology of endometriosis is not understood, it is known that the ectopic endometrial tissue is sensitive to the production of sex steroid hormones during the menstrual cycle. Endometriosis is rare before menarche and develops usually during the reproductive years. The importance of ovarian steroids in the development and maintenance of endometriosis is also demonstrated by the usual curative effect of oophorectomy and the spontaneous regression of the disease at menopause. Experimentally implanted endometriosis in castrated monkeys requires sex steroids for its maintenance [1]. Preview Unable to display preview. Download preview PDF. Similar content being viewed by others

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Reversible bone loss is induced by GnRH agonist. 68th Annual Meeting of the Endocrine Society, Anaheim, California, Abstract Matta, WH, Shaw, RW, Hesp, R, and Katz, D (1987). Hypogonadism induced by luteinizing hormone releasing hormone agonist analogues: effects on bone density in premenopausal women. Brit Med J, 294. 1523 Devogelaer, JP, DeDeuxchaisnes, CN, Donnez, J, and Thomas, K (1987). LHRH analogues and bone loss. The Lancet, I 1498 Author information Authors and Affiliations Consortia Editor information Editors and Affiliations Rights and permissions Copyright information © 1990 Springer Science+Business Media Dordrecht About this chapter Cite this chapter Lemay, A., HRPI buserelin protocol 310 study group. (1990). Comparison of Buserelin to Danazol Therapy in Endometriosis. In: Vickery, B.H., Lunenfeld, B. (eds) GnRH Analogues in Cancer and Human Reproduction. Springer, Dordrecht. https://doi.org/10.1007/978-94-009-2169-6_3 Download citation DOI: https://doi.org/10.1007/978-94-009-2169-6_3 Publisher Name: Springer, Dordrecht Print ISBN: 978-94-010-7474-2 Online ISBN: 978-94-009-2169-6 eBook Packages: Springer Book Archive

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