Aberrantly expressed long noncoding RNAs in the eutopic endometria of patients with uterine adenomyosis
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This study identified specific long noncoding RNAs that are abnormally expressed in the uterine endometria of patients diagnosed with adenomyosis.
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References (30)
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Cited by (16)
- The Art of Managing Infertile Patients with Adenomyosis 2024
- Bioinformatic analysis and machine learning to identify the diagnostic biomarkers and immune infiltration in adenomyosis 2023
- Bromocriptine inhibits proliferation in the endometrium from women with adenomyosis 2023
- MIR503HG silencing promotes endometrial stromal cell progression and metastasis and suppresses apoptosis in adenomyosis by activating the Wnt/β‑catenin pathway via targeting miR‑191 2023
- Role of lncRNA TUG1 in Adenomyosis and its Regulatory Mechanism in Endometrial Epithelial Cell Functions 2022
- Adenomyosis and fertility: a modern view of the problem. A literature review 2022
- Transcriptomics of receptive endometrium in women with sonographic features of adenomyosis 2022
- Physiopathologie de l’adénomyose : une énigme non résolue 2021
- Endometrial causes of recurrent pregnancy losses: endometriosis, adenomyosis, and chronic endometritis 2021
- Preliminary functional inquiry of lncRNA ENST00000433673 in embryo implantation using bioinformatics analysis 2019
- Bioinformatics strategy for the screening of key genes to differentiate adenomyosis from endometriosis (Review) 2019
- Uterine polyps, adenomyosis, leiomyomas, and endometrial receptivity 2019
- The Association of Sonographic Evidence of Adenomyosis with Severe Endometriosis and Gene Expression in Eutopic Endometrium 2018
- The Molecular Basis of Adenomyosis Development 2018
- Pathogenesis of adenomyosis: an update on molecular mechanisms 2017
- Role of benign ovarian cysts in the development of adenomyosis 2016
Source provenance
- europepmc
- last seen: 2026-06-04T01:30:01.192114+00:00
- openalex
- last seen: 2026-06-04T00:00:01.174412+00:00
- pubmed
- last seen: 2026-05-13T22:21:13.485820+00:00
License: CC0
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