Additional file 7 of The IL-33-ST2 axis plays a vital role in endometriosis via promoting epithelial–mesenchymal transition by phosphorylating β-catenin
This study investigated the autocrine effect of IL-33 on stromal cells, finding it influences α-SMA expression, cell viability, and migration in eutopic and ectopic endometrial cells from endometriosis patients.
One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works
Abstract
My notes (saved in your browser only)
Condition tags
Citation neighborhood (2-hop)
Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. Outer rings show 2-hop neighbours — papers reached through the immediate citers/citees. [ collapse to 1-hop ]
References (60)
- A high level of TGF-B1 promotes endometriosis development via cell migration, adhesiveness, colonization, and invasiveness† via openalex
- A long-acting anti-IL-8 antibody improves inflammation and fibrosis in endometriosis via openalex
- CD4+Foxp3+ regulatory T cell differentiation mediated by endometrial stromal cell-derived TECK promotes the growth and invasion of endometriotic lesions via openalex
- Circulating estradiol and its biologically active metabolites in endometriosis and in relation to pain symptoms via openalex
- Distinct subtypes of endometriosis identified based on stromal-immune microenvironment and gene expression: implications for hormone therapy via openalex
- Epithelial–Mesenchymal Transition in Endometriosis—When Does It Happen? via openalex
- E<sub>2</sub>‐mediated EMT by activation of β‐catenin/Snail signalling during the development of ovarian endometriosis via openalex
- Expression is Down-Regulated by Oxidative Stress in Endometriosis and Endometriosis-Associated Ovarian Cancer via openalex
- IL-27 triggers IL-10 production in Th17 cells via a c-Maf/RORγt/Blimp-1 signal to promote the progression of endometriosis via openalex
- Increased Expression of TGF-β1 Contributes to the Downregulation of Progesterone Receptor Expression in the Eutopic Endometrium of Infertile Women with Minimal/Mild Endometriosis via openalex
- Interleukin-33 modulates inflammation in endometriosis via openalex
- Interleukin-33 promotes invasiveness of human ovarian endometriotic stromal cells through the ST2/MAPK/MMP-9 pathway activated by 17β-estradiol via openalex
- Local and systemic levels of cytokines and danger signals in endometriosis-affected women via openalex
- Macrophages originated IL-33/ST2 inhibits ferroptosis in endometriosis via the ATF3/SLC7A11 axis via openalex
- New Therapeutics in Endometriosis: A Review of Hormonal, Non-Hormonal, and Non-Coding RNA Treatments via openalex
- NLRP3 Inflammasome Activation of Mast Cells by Estrogen via the Nuclear-Initiated Signaling Pathway Contributes to the Development of Endometriosis via openalex
- Oestrogen-induced epithelial-mesenchymal transition (EMT) in endometriosis: Aetiology of vaginal agenesis in Mayer-Rokitansky-Küster-Hauser (MRKH) syndrome via openalex
- Oxidative Stress as a Potential Underlying Cause of Minimal and Mild Endometriosis-Related Infertility via openalex
- Rethinking mechanisms, diagnosis and management of endometriosis via openalex
- Serum and peritoneal interleukin-33 levels are elevated in deeply infiltrating endometriosis via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
- (no identifier) via openalex
Source provenance
- openalex
- last seen: 2026-05-13T20:12:55.465734+00:00