Konstantin J Dedes

No ORCID on file · 6 papers in corpus · active 2012-2013

Study types

  • article 5
  • review 1

Condition tags

  • endometriosis 6
review 2013
ARID1A mutations and PI3K/AKT pathway alterations in endometriosis and endometriosis-associated ovarian carcinomas
International journal of molecular sciences ·doi:10.3390/ijms140918824

Endometriosis is a common gynecological disease affecting 6%-10% of women of reproductive age and is characterized by the presence of endometrial-like tissue in localizations outside of the uterine cavity as, e.g., endometriotic ovarian cys…

article 2013
Histone deacetylase inhibitors down-regulate G-protein-coupled estrogen receptor and the GPER-antagonist G-15 inhibits proliferation in endometriotic cells
Fertility and sterility ·doi:10.1016/j.fertnstert.2013.05.008
article 2013
receptor (GPER) in endometriosis: a tissue
article 2013
www.mdpi.com/journal/ijms Review ARID1A Mutations and PI3K/AKT Pathway Alterations in

Abstract: Endometriosis is a common gynecological disease affecting 6%–10 % of women of reproductive age and is characterized by the presence of endometrial-like tissue in localizations outside of the uterine cavity as, e.g., endometriotic …

article 2012
Expression of the G protein-coupled estrogen receptor (GPER) in endometriosis: a tissue microarray study
Reproductive biology and endocrinology : RB&E ·doi:10.1186/1477-7827-10-30

BACKGROUND: The G protein-coupled estrogen receptor (GPER) is thought to be involved in non-genomic estrogen responses as well as processes such as cell proliferation and migration. In this study, we analyzed GPER expression patterns from e…

article 2012
Loss of ARID1A/BAF250a-expression in endometriosis: a biomarker for risk of carcinogenic transformation?
Modern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc ·doi:10.1038/modpathol.2011.217