Schmutzler AG

No ORCID on file · 5 papers in corpus · active 2004-2011

Study types

  • article 5

Condition tags

  • endometriosis 5
article 2011
BioImpacts : BI ·doi:10.5681/bi.2011.017

INTRODUCTION: In a cytological analysis of endometriotic lesions neither granulocytes nor cytotoxic T-cells appear in an appreciable number. Based on this observation we aimed to know, whether programmed cell death plays an essential role i…

article 2008
Fertility and sterility ·doi:10.1016/j.fertnstert.2007.06.060

ObjectiveTo evaluate the concentration and distribution pattern of interleukin-6-producing cells in ectopic and eutopic endometrium.DesignCollection of both tissues at the proliferative cycle phase.SettingA university endocrinology and lapa…

article 2007
Journal of assisted reproduction and genetics ·doi:10.1007/s10815-007-9116-y

ProblemAs recent studies have suggested abnormalities in the regulation of specific genes in the development of endometriosis, we investigated differentially expressed genes in endometriosis compared to endometrium.Method of studyGene expre…

article 2005
Human Reproduction ·doi:10.1093/humrep/dei071

BACKGROUND: We investigated granulocyte colony-stimulating factor (G-CSF) in human reproduction. METHODS: From a total sample of 93 patients, we analysed in group 1 (n = 82) the level of G-CSF and estradiol (E(2)) in serum and follicular fl…

article 2004
American journal of reproductive immunology (New York, N.Y. : 1989) ·doi:10.1111/j.1600-0897.2004.00228.x

PROBLEM: The aim of this paper is to provide further evidence that the dystopic proliferation of endometriotic epithelia is caused by the stimulation of peritoneal macrophages. It is essential to show that endometriotic epithelial cells exp…