studyfor the treatment of endometriosis: an in vitro B inhibitor BAY κ Application of the nuclear factor-
OA: closed
CC0
Limited metadata. Only one source feed has indexed
this record so far — no abstract, full text, or open-access copy is
available through Endo Lab. The
publisher
is the canonical location for the actual content. If you have institutional
access, use "Find at my library".
AI-generated summary
This paper investigates the in vitro application of the nuclear factor-kappa B inhibitor BAY 11-7082 for the treatment of endometriosis.
One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works
My notes (saved in your browser only)
Condition tags
Citation neighborhood (2-hop)
Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. Outer rings show 2-hop neighbours — papers reached through the immediate citers/citees. [ collapse to 1-hop ]
References (33)
- Apoptosis and bcl-2 expression in normal human endometrium, endometriosis and adenomyosis via openalex
- Bufalin induces apoptosis and the G0/G1 cell cycle arrest of endometriotic stromal cells: a promising agent for the treatment of endometriosis via openalex
- Down-regulation of interleukin-1 receptor type 1 expression causes the dysregulated expression of CXC chemokines in endometriotic stromal cells: a possible mechanism for the altered immunological functions in endometriosis via openalex
- Drug-induced apoptosis was markedly attenuated in endometriotic stromal cells via openalex
- Effects of triptorelin versus placebo on the symptoms of endometriosis via openalex
- Endometriotic cells are resistant to interferon-gamma-induced cell growth inhibition and apoptosis: a possible mechanism involved in the pathogenesis of endometriosis via openalex
- Lipopolysaccharide-promoted proliferation of endometriotic stromal cells via induction of tumor necrosis factor alpha and interleukin-8 expression via openalex
- Long-term progestin treatment inhibits RANTES (regulated on activation, normal T cell expressed and secreted) gene expression in human endometrial stromal cells via openalex
- New considerations for the pathogenesis of endometriosis via openalex
- Possible pathophysiological roles of mitogen-activated protein kinases (MAPKs) in endometriosis via openalex
- Role of cytokines in endometriosis via openalex
- Spontaneous apoptosis of endometrial tissue is impaired in women with endometriosis via openalex
- Sulindac suppresses nuclear factor-kappaB activation and RANTES gene and protein expression in endometrial stromal cells from women with endometriosis via openalex
- Tumor necrosis factor-alpha-induced interleukin-8 (IL-8) expression in endometriotic stromal cells, probably through nuclear factor-kappa B activation: gonadotropin-releasing hormone agonist treatment reduced IL-8 expression via openalex
- W2151892297 via openalex
- W2171865516 via openalex
- W2288500425 via openalex
- W2316386966 via openalex
- W1481925502 via openalex
- W2331176038 via openalex
- W1492606093 via openalex
- W1520994322 via openalex
- W1888783223 via openalex
- W1981834168 via openalex
- W1990065094 via openalex
- W1995335649 via openalex
- W2006456625 via openalex
- W2018011543 via openalex
- W2028802255 via openalex
- W2076485183 via openalex
- W2090850983 via openalex
- W2117021879 via openalex
- W2126729416 via openalex
Source provenance
- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00
License: CC0
· commercial use OK