Endometriosis: A Review of Systematic Reviews and Meta-Analysis

Mehmet Yılmaz
In: MAS Journal Of Applied Sciences · 2022 · vol. 7(11) · doi:10.52520/masjaps.206 · W4214756239
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⚙ AI-generated summary by claude@2026-06, 2026-06-09 ⓘ

This review consolidates systematic reviews and meta-analyses published after 2015 on endometriosis, a common disease often diagnosed late, dependent on estrogen, and with incompletely understood pathogenesis involving the immune system.

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Abstract

Endometriosis is a common disease with typically delayed diagnosis. Its prevalence is high among adolescents with pelvic pain symptom. Deposits of tissue outside uterine cavity depend on oestrogen. Progestins and oral contraceptives are not successful 1/3 of symptomatic women worldwide. Pathogenesis of endometriosis is not well understood but immune system plays a role in its pathophysiology. The aetiology of endometriosis remains largely unknown. Here in this review, reader may find a review of systematic reviews and meta-analysis published after 2015 on endometriosis.
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Abstract

Endometriosis is a common disease with typically delayed diagnosis. Its prevalence is high amongadolescents with pelvic pain symptom. Deposits of tissue outside uterine cavity depend on oestrogen.Progestins and oral contraceptives are not successful 1/3 of symptomatic women worldwide. Pathogenesisof endometriosis is not well understood but immune system plays a role in its pathophysiology. Theaetiology of endometriosis remains largely unknown. Here in this review, reader may find a review ofsystematic reviews and meta-analysis published after 2015 on endometriosis.

Keywords

Endometriosis,meta-analysis,systematic review How to Cite YILMAZ, M. . (2022). Endometriosis: A Review of Systematic Reviews and Meta-Analysis. MAS Journal of Applied Sciences, 7(1), 73–80. https://doi.org/10.52520/masjaps.206

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Condition tags

endometriosis

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (21)

Source provenance

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