Dinitrosyl iron complexes with glutathione suppress experimental endometriosis in rats
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Dinitrosyl iron complexes with glutathione were found to suppress experimental endometriosis in a rat model.
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References (39)
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Cited by (11)
- The role of macrophage polarization in the development of endometriosis and the therapeutic potential of its modulation 2025
- Experimental modeling, prevention, and treatment of endometriosis-associated adhesion formation in NIH/3T3 fibroblast cell line using bovhyaluronidase azoximer (in vitro) 2024
- Modern view on the experimental creation of endometriosis in animal models (own data) 2024
- Oxidative stress, ferroptosis, somatic mutations, antioxidant therapy, and endometriosis: a new perspective on the issue 2024
- The role of ferroptosis in the pathogenesis and progression of endometriosis. History of the question and current evidence 2023
- The Inhibiting Effect of Dinitrosyl Iron Complexes with Thiol-containing Ligands on the Growth of Endometrioid Tumours in Rats with Experimental Endometriosis 2019
- An antinitrosative system as a factor in malignant tumor resistance to the cytotoxic effect of nitrogen monoxide 2015
- Dinitrosyl Iron Complexes with Glutathione Suppress Surgically Induced Experimental Endometriosis in Rats 2015
- Antitumor activity of dinitrosyl iron complexes with glutathione 2014
- The effect of dinitrosyl iron complexes with glutathione and S-nitrosoglutathione on the development of experimental endometriosis in rats: A comparative studies 2014
- Physicochemistry of dinitrosyl iron complexes with thiolate ligands underlying their beneficial effect in endometriosis 2014
Source provenance
- europepmc
- last seen: 2026-06-13T06:22:48.782012+00:00
- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00
- pubmed
- last seen: 2026-05-13T22:18:40.923139+00:00
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