Chronic pelvic pain in women: role of the nervous system

Wayne R. Gillett, David S. Jones
In: Expert Review of Obstetrics & Gynecology · 2009 · vol. 4(2) , pp. 149–163 · doi:10.1586/17474108.4.2.149 · W2135404229
Review OA: closed CC0 ⤵ 4 in-corpus citations
View on OpenAlex View at publisher
⚙ AI-generated summary by gemini-2.5-flash-lite, 2026-06-08 ⓘ

This review correlates knowledge on nervous system mechanisms, including peripheral and central sensitization, that contribute to chronic pelvic pain in women, highlighting a lack of evidence-based management.

One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works

Abstract

There is increasing recognition that mechanisms in the nervous system common to other persistent pain conditions and inflammation of neurogenic origin play a major role in chronic pelvic pain. For either somatic or visceral pain, an understanding of the processes of peripheral and central sensitization mechanisms, common to both acute and persistent pain, should help explain difficult pain conditions and open new therapeutic options. This review aims to correlate knowledge regarding persistent pain causation and pain conditions in gynecology. Although there is emerging evidence about the pathophysiology and management of persistent pain, there is still a dearth of evidence-based practice for chronic pelvic pain in women.

My notes (saved in your browser only)

Condition tags

chronic_pelvic_pain

Citation neighborhood (2-hop)

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. Outer rings show 2-hop neighbours — papers reached through the immediate citers/citees. [ collapse to 1-hop ]

References (100)

Cited by (4)

Source provenance

openalex
last seen: 2026-06-10T17:14:06.276822+00:00
unpaywall
last seen: 2026-10-11T06:28:45.473802+00:00
License: CC0 · commercial use OK